This product is a peripheral dopamine receptor antagonist, which can promote the peristalsis and tension of the upper gastrointestinal tract to return to normal, promote gastric emptying, increase the movement of the gastric antrum and duodenum, coordinate the contraction of the pylorus, and also enhance the peristalsis of the esophagus and the tension of the lower esophageal sphincter, inhibit nausea and vomiting, and do not affect the secretion of gastric juice. This product is not easy to pass through the blood-brain barrier and has almost no antagonistic effect on dopamine receptors in the brain, so it generally has no adverse reactions to the mental and central nervous systems.

This product is a peripheral dopamine receptor antagonist, which can promote the peristalsis and tension of the upper gastrointestinal tract to return to normal, promote gastric emptying, increase the movement of the gastric antrum and duodenum, coordinate the contraction of the pylorus, and also enhance the peristalsis of the esophagus and the tension of the lower esophageal sphincter, inhibit nausea and vomiting, and do not affect the secretion of gastric juice. This product is not easy to pass through the blood-brain barrier and has almost no antagonistic effect on dopamine receptors in the brain. Therefore, it generally has no adverse reactions to the mental and central nervous systems.

This product is a peripheral dopamine receptor antagonist, which can promote the peristalsis and tension of the upper gastrointestinal tract to return to normal, promote gastric emptying, increase the movement of the gastric antrum and duodenum, coordinate the contraction of the pylorus, and also enhance the peristalsis of the esophagus and the tension of the lower esophageal sphincter, inhibit nausea and vomiting, and do not affect the secretion of gastric juice. This product is not easy to pass through the blood-brain barrier and has almost no antagonistic effect on dopamine receptors in the brain. Therefore, it generally has no adverse reactions to the mental and central nervous systems.

This product can reduce the level of DHBV-DNA in the serum of ducks infected with duck hepatitis B virus (DHBV); it has a protective effect on toxic liver damage in mice caused by CCl4 and D-galactosamine.

Increase the synthesis of GABA and reduce the degradation of GABA, thereby increasing the concentration of the inhibitory neurotransmitter g-aminobutyric acid (GABA), reducing the excitability of neurons and inhibiting seizures. In electrophysiological experiments, this product can produce an inhibitory effect on Na channels similar to that of phenytoin. It is harmful to the liver.