Neutralizes gastric acid, reduces the irritation and corrosion of gastric acid on the ulcer surface, improves the resistance of gastric mucosa, and enhances digestive function; has anti-inflammatory and Helicobacter pylori inhibitory effects

Neutralize gastric acid, reduce the irritation and corrosion of gastric acid on the ulcer surface, and facilitate the healing of the ulcer surface

Relieve pain caused by smooth muscle spasm, improve gastric mucosal resistance, and enhance digestive function

Improve gastric mucosal resistance and enhance digestive function

Relieve pain caused by smooth muscle spasm

Improve gastric mucosal resistance and enhance digestive function

A cool feeling

Has a certain central sedative effect

Camphor has a cooling effect, and chloral hydrate has a certain central sedative effect. This product has a mild analgesic effect on neuralgia when used externally.

Acts on dihydrofolate synthase, interfering with each step of folic acid synthesis

Acts on the second step of folate anabolism, selectively inhibiting dihydrofolate reductase

Sulfadimethoxazole (SMZ) and trimethoprim (TMP) have synergistic antibacterial or bactericidal effects because sulfa acts on dihydrofolate synthase, interfering with every step of folic acid synthesis, while trimethoprim acts on the second step of folic acid anabolism, selectively resisting dihydrofolate reductase. Therefore, the combined use of the two can doubly block the bacterial folate metabolism.

Sulfadimethoxazole (SMZ) and trimethoprim (TMP) have synergistic antibacterial or bactericidal effects because sulfa acts on dihydrofolate synthase, interfering with every step of folic acid synthesis, while trimethoprim acts on the second step of folic acid anabolism, selectively resisting dihydrofolate reductase. Therefore, the combined use of the two can doubly block the bacterial folate metabolism.

This product is a benzimidazole derivative, which is rapidly metabolized into sulfoxide, sulfone alcohol and 2-amine sulfone alcohol in the body. It selectively and irreversibly inhibits the polymerization of the cytoplasmic microtubule system of the parasite's intestinal wall cells, blocks its absorption of various nutrients and glucose, leads to the depletion of endogenous glycogen in the worm, and inhibits the fumarate reductase system, preventing the production of adenosine triphosphate, making it impossible for the worm to survive and reproduce. Similar to mebendazole, this product can also cause the degeneration of the cytoplasmic microtubules of the worm's intestinal cells, and bind to its microtubule protein, causing intracellular transport blockage, resulting in the accumulation of Golgi endocrine granules, the gradual dissolution of the cytoplasm, and the complete degeneration of the absorbing cells, causing the death of the worm. This product has the effect of completely killing hookworm eggs and whipworm eggs and partially killing ascarid eggs. In addition to killing and expelling various nematodes parasitic in animals, it also has a significant killing and expelling effect on tapeworms and cysticerci.