This product is a kind of thrombin extracted from the venom of the white-browed agkistrodon acutus in Changbai Mountain. It contains thrombin-like enzymes and thrombin-like kinases. The two types of enzymes are similar substrates. In the presence of Ca2+, they can activate factors V, VII and VIII and stimulate platelet aggregation; in the presence of platelet factor III, thrombin-like kinases can promote the conversion of prothrombin into thrombin, activate factor V, and affect factor X. Animal test results show that this product has a procoagulant effect at low doses and an anticoagulant effect at high doses.

This product contains thrombin-like enzymes and thrombin-like kinases, which can activate factors V, VII and VIII in the presence of Ca2+, and stimulate platelet aggregation; thrombin-like kinases can promote the conversion of prothrombin into thrombin in the presence of platelet factor III, and can also activate factor V and affect factor X. It has a procoagulant effect at low doses and an anticoagulant effect at high doses.

This product is a kind of thrombin extracted from the venom of the white-browed agkistrodon acutus in Changbai Mountain. It contains thrombin-like enzymes and thrombin-like kinases. The two enzymes are similar substrates. In the presence of Ca2, they can activate factors V, VII and VIII and stimulate platelet aggregation; in the presence of platelet factor III, thrombin-like kinases can promote the conversion of prothrombin into thrombin, activate factor V, and affect factor X. Animal test results show that this product has a procoagulant effect at low doses and an anticoagulant effect at high doses.

No specific pharmacological effects mentioned

This product is a new type of selective anticholinergic drug that can enter the brain through the blood-brain barrier. It can block the stimulatory effect of acetylcholine on muscarinic receptors (M receptors) and nicotinic receptors (N receptors) in the brain; therefore, it can better antagonize the central poisoning symptoms caused by organophosphorus poisons (pesticides), such as convulsions, central respiratory and circulatory failure, and irritability. At the same time, it also has a strong blocking effect on the stimulatory effect of acetylcholine on M receptors in the periphery; therefore, it can better antagonize the muscarinic poisoning symptoms caused by organophosphorus poisons (pesticides), such as bronchial smooth muscle spasm and increased secretion, sweating, dilation, miosis, and gastrointestinal smooth muscle spasm or contraction. It can also increase respiratory rate and respiratory flow, but since this product has no obvious effect on M2 receptors, it has no obvious effect on heart rate; it has no obvious antagonistic effect on peripheral N receptors.