This product can directly penetrate the cell membrane into the body cells, activate pyruvate oxidase, so that cells in a low-energy and hypoxic state can continue to metabolize smoothly, and participate in the energy metabolism and protein synthesis of the human body.

This product is an endogenous coenzyme B12, which participates in the one-carbon unit cycle and plays an important role in the transmethylation reaction of methionine synthesis from homocysteine. Animal experiments have found that this product is easier to enter neuronal organelles than cyanocobalamin, participates in the synthesis of thymidine nucleoside in brain cells and spinal cord neurons, promotes the utilization of folic acid and nucleic acid metabolism, and has a stronger effect on promoting nucleic acid and protein synthesis than cyanocobalamin. This product can promote axonal transport function and axonal regeneration, normalize the transport of axonal skeleton proteins in the sciatic nerve of diabetic rats induced by streptozotocin, and has an inhibitory effect on drug-induced neurodegeneration, such as neurodegeneration caused by adriamycin, acrylamide, and vincristine, and neurological diseases in spontaneously hypertensive rats. In rat tissue culture, it was found that this product can promote lecithin synthesis and neuronal myelination. This product can restore delayed synaptic transmission and neurotransmitter reduction to normal, restore end plate potential induction by increasing nerve fiber excitability, and restore acetylcholine in the brain of rats fed with choline-deficient feed to normal levels.

This product is an anti-anemia drug. Vitamin B12 participates in methyl conversion and folic acid metabolism in the body, and promotes the conversion of 5-methyltetrahydrofolate into tetrahydrofolate. When it is deficient, it leads to DNA synthesis disorders and affects the maturation of red blood cells. This product also promotes the conversion of methylmalonic acid into succinic acid and participates in the tricarboxylic acid cycle. This effect is related to the synthesis of nerve myelin lipids and the maintenance of the integrity of myelinated nerve fiber function. The nerve damage caused by vitamin B12 deficiency may be related to this.

It has a good antibacterial effect on human tuberculosis bacteria, and its bactericidal effect is strongest at pH 5-5.5. It is currently the best bactericidal drug for tuberculosis bacteria that grow slowly in phagocytes in an acidic environment. The antibacterial concentration in vivo is 12.5 mu;g/ml, and 50 mu;g/ml can kill tuberculosis bacteria. The concentration that inhibits tuberculosis bacteria inside cells is 10 times lower than that outside cells, and it has almost no antibacterial effect in neutral and alkaline environments. The mechanism of action may be related to pyrazinoic acid, which interferes with dehydrogenase by replacing nicotinamide, prevents dehydrogenation, hinders the use of oxygen by tuberculosis bacteria, affects the normal metabolism of bacteria and causes death.

This product is a derivative of cantharidin, which inhibits DNA synthesis of cancer cells and interferes with the M phase of cell division. Animal experiments show that this product has a certain inhibitory effect on mouse liver cancer and ascites cancer. At a blood concentration of 16~18.5mg/ml, it can prolong the survival time of mice by 31%~103%. This product has a certain effect on the body's immune function. Intravenous injection of methylcantharidin can enhance the phagocytosis of macrophages in animals and inhibit the production of antibodies.