Inhibits uric acid synthesis. By inhibiting xanthine oxidase, it blocks the metabolism of hypoxanthine and xanthine into uric acid, reduces uric acid production, lowers uric acid levels in blood and urine, prevents uric acid crystal deposition, and helps to redissolve uric acid crystals in the tissues of gout patients; at the same time, it inhibits new purine synthesis in the body. Blood uric acid concentration begins to decrease 24 hours after oral administration, and the decrease is most obvious in 2 to 4 weeks.

Some patients may experience gastrointestinal reactions, heart, liver, and kidney damage due to drug overdose. The dosage may be reduced or symptomatic treatment may be given as appropriate.

In case of drug overdose, some patients may experience gastrointestinal reactions and damage to the heart, liver, and kidneys. The dosage may be reduced or symptomatic treatment may be given as appropriate.

1. 5-HT3 receptor antagonism: In the 5-hydroxytryptamine-induced guinea pig isolated colon contraction experiment and rat and ferret heart rate slowing reflex (Bezold-Jarisch reflex) experiment, it showed 5-HT3 receptor antagonism. 2. Anti-malignant tumor drug-induced vomiting inhibition: The vomiting of ferrets induced by cisplatin was inhibited by giving this product before or after the initial vomiting. Cisplatin and other anti-malignant tumor drugs can free 5-hydroxytryptamine from the chromaffin cells in the digestive tract, and 5-hydroxytryptamine binds to the 5-HT3 receptors present in the gastrointestinal mucosa and the vagus nerve endings.