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Founded in:
2001-01-16 -
Country:
China -
Address:
No. 9 Xianlin Avenue, Qixia District, Nanjing -
Tax NO.:
91320192726063334T -
Registered Funds:
50 million yuan -
Website:
-
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Tranexamic Acid |
Competitively inhibit the adsorption of plasminogen on fibrin, prevent its activation, protect fibrin from being degraded and dissolved by plasmin, and achieve hemostatic effect; directly inhibit plasmin activity, reduce the effect of plasmin activating complement, and prevent the occurrence of hereditary angioedema.
More
Competitively inhibit the adsorption of plasminogen on fibrin, prevent its activation, protect fibrin from being degraded and dissolved by plasmin, and achieve hemostatic effect; directly inhibit plasmin activity, reduce the effect of plasmin activating complement, and prevent the occurrence of hereditary angioedema. |
1197-18-8 | 30 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Tranexamic Acid |
Competitively inhibit the adsorption of plasminogen on fibrin, prevent its activation, protect fibrin from being degraded and dissolved by plasmin, and achieve hemostatic effect; directly inhibit plasmin activity, reduce the effect of plasmin activating complement, and prevent the occurrence of hereditary angioedema.
More
Competitively inhibit the adsorption of plasminogen on fibrin, prevent its activation, protect fibrin from being degraded and dissolved by plasmin, and achieve hemostatic effect; directly inhibit plasmin activity, reduce the effect of plasmin activating complement, and prevent the occurrence of hereditary angioedema. |
1197-18-8 | 30 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Tranexamic Acid |
It competitively inhibits the adsorption of plasminogen on fibrin, prevents its activation, and protects fibrin from being degraded and dissolved by plasmin, thereby achieving a hemostatic effect. It can also directly inhibit the activity of plasmin, reduce the effect of plasmin in activating complement, and prevent the occurrence of hereditary angioedema.
More
It competitively inhibits the adsorption of plasminogen on fibrin, prevents its activation, and protects fibrin from being degraded and dissolved by plasmin, thereby achieving a hemostatic effect. It can also directly inhibit the activity of plasmin, reduce the effect of plasmin in activating complement, and prevent the occurrence of hereditary angioedema. |
1197-18-8 | 30 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Cytidine 5'-triphosphate disodium salt |
Coenzyme drugs are nucleotide derivatives that participate in the synthesis and metabolism of phospholipids and nucleic acids in the body. They are intermediate products and energy sources for brain phospholipid synthesis and nucleic acid metabolism.
More
Coenzyme drugs are nucleotide derivatives that participate in the synthesis and metabolism of phospholipids and nucleic acids in the body. They are intermediate products and energy sources for brain phospholipid synthesis and nucleic acid metabolism. |
36051-68-0 | 10 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Mecobalamin |
Methylcobalamin is an endogenous coenzyme B12 that plays an important role as a coenzyme of methionine synthase in the transmethylation reaction of methionine synthesized from homocysteine. It is easily transferred to the organelles of nerve cells, thereby promoting the synthesis of nucleic acids and proteins. It promotes axonal transport and axonal regeneration. It is effective against sciatica neuropathy in diabetic rats caused by streptozotocin.
More
Methylcobalamin is an endogenous coenzyme B12 that plays an important role as a coenzyme of methionine synthase in the transmethylation reaction of methionine synthesized from homocysteine. It is easily transferred to the organelles of nerve cells, thereby promoting the synthesis of nucleic acids and proteins. It promotes axonal transport and axonal regeneration. It is effective against sciatica neuropathy in diabetic rats caused by streptozotocin. |
13422-55-4 | 28 |