-
Founded in:
2000-04-24 -
Country:
China -
Address:
No. 180, Yixing Avenue, Yiling District, Yichang City, Hubei Province -
Tax NO.:
914205066764994030 -
Registered Funds:
73.460277 yuan -
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Omeprazole sodium |
This product is a proton pump inhibitor of gastric parietal cells, which can specifically inhibit the secretory microtubules and H, K-ATPases on the tubular vesicles in the cytoplasm of the parietal cell apical membrane, thereby effectively inhibiting the secretion of gastric acid. It can not only non-competitively inhibit the gastric acid secretion caused by gastrin, histamine, choline, food, and stimulation of the vagus nerve, but also inhibit part of the basic gastric acid secretion that is not affected by choline or H2 receptor blockers. It also has a strong and lasting inhibitory effect on the gastric acid secretion caused by dibutyl cyclic adenosine monophosphate (DCAMP) stimulation that cannot be inhibited by H2 receptor antagonists. In addition, this product also has an inhibitory effect on the secretion of pepsin, has no obvious change in gastric mucosal blood flow, and does not affect body temperature, gastric cavity temperature, arterial blood pressure, venous hemoglobin, arterial oxygen partial pressure, carbon dioxide partial pressure and arterial blood pH.
More
This product is a proton pump inhibitor of gastric parietal cells, which can specifically inhibit the secretory microtubules and H, K-ATPases on the tubular vesicles in the cytoplasm of the parietal cell apical membrane, thereby effectively inhibiting the secretion of gastric acid. It can not only non-competitively inhibit the gastric acid secretion caused by gastrin, histamine, choline, food, and stimulation of the vagus nerve, but also inhibit part of the basic gastric acid secretion that is not affected by choline or H2 receptor blockers. It also has a strong and lasting inhibitory effect on the gastric acid secretion caused by dibutyl cyclic adenosine monophosphate (DCAMP) stimulation that cannot be inhibited by H2 receptor antagonists. In addition, this product also has an inhibitory effect on the secretion of pepsin, has no obvious change in gastric mucosal blood flow, and does not affect body temperature, gastric cavity temperature, arterial blood pressure, venous hemoglobin, arterial oxygen partial pressure, carbon dioxide partial pressure and arterial blood pH. |
95510-70-6 | 24 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Sodium ferulic |
This product is a non-peptide endothelin receptor antagonist, which can antagonize endothelin-induced vasoconstriction, blood pressure increase and vascular smooth muscle cell proliferation, reduce vascular endothelial damage; increase NO synthesis, relax vascular smooth muscle; inhibit platelet aggregation, anti-coagulation, and improve blood rheology characteristics. This product can also inhibit cholesterol synthesis, lower blood lipids, scavenge free radicals, prevent lipid peroxidation damage; affect complement, enhance immune function, and have certain analgesic and antispasmodic effects.
More
This product is a non-peptide endothelin receptor antagonist, which can antagonize endothelin-induced vasoconstriction, blood pressure increase and vascular smooth muscle cell proliferation, reduce vascular endothelial damage; increase NO synthesis, relax vascular smooth muscle; inhibit platelet aggregation, anti-coagulation, and improve blood rheology characteristics. This product can also inhibit cholesterol synthesis, lower blood lipids, scavenge free radicals, prevent lipid peroxidation damage; affect complement, enhance immune function, and have certain analgesic and antispasmodic effects. |
24276-84-4 | 8 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Coenzyme A |
A coenzyme for acetylation reactions in the body. It participates in acetylation reactions in the body and plays an important role in the metabolism of sugar, fat and protein, such as the tricarboxylic acid cycle, liver glycogen accumulation, acetylcholine synthesis, lowering cholesterol, regulating blood lipid content and synthesizing steroid substances, which are all closely related to this product.
More
A coenzyme for acetylation reactions in the body. It participates in acetylation reactions in the body and plays an important role in the metabolism of sugar, fat and protein, such as the tricarboxylic acid cycle, liver glycogen accumulation, acetylcholine synthesis, lowering cholesterol, regulating blood lipid content and synthesizing steroid substances, which are all closely related to this product. |
85-61-0 | 4 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Sodium aescinate 98% |
It can help the body increase the plasma concentration of ACTH and cortisone, promote the secretion of PGF2α by the blood vessel wall, remove free radicals in the body, resist inflammation and exudation, increase venous tension, speed up venous blood flow, promote lymphatic return, improve blood circulation and microcirculation, and protect the blood vessel wall.
More
It can help the body increase the plasma concentration of ACTH and cortisone, promote the secretion of PGF2α by the blood vessel wall, remove free radicals in the body, resist inflammation and exudation, increase venous tension, speed up venous blood flow, promote lymphatic return, improve blood circulation and microcirculation, and protect the blood vessel wall. |
0 | ||
| Sodium aescinate B |
It can help the body increase the plasma concentration of ACTH and cortisone, promote the secretion of PGF2α by the blood vessel wall, remove free radicals in the body, resist inflammation and exudation, increase venous tension, speed up venous blood flow, promote lymphatic return, improve blood circulation and microcirculation, and protect the blood vessel wall.
More
It can help the body increase the plasma concentration of ACTH and cortisone, promote the secretion of PGF2α by the blood vessel wall, remove free radicals in the body, resist inflammation and exudation, increase venous tension, speed up venous blood flow, promote lymphatic return, improve blood circulation and microcirculation, and protect the blood vessel wall. |
0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Clotrimazole |
1. Inhibit plasminogen activators, so that plasminogen cannot be activated into plasmin, thereby inhibiting the dissolution of fibrin and producing a hemostatic effect; 2. Promote the release of active substances by platelets, enhance platelet aggregation and adhesion, shorten coagulation time, and produce a hemostatic effect; 3. Enhance capillary resistance, reduce capillary permeability, and thus reduce bleeding.
More
1. Inhibit plasminogen activators, so that plasminogen cannot be activated into plasmin, thereby inhibiting the dissolution of fibrin and producing a hemostatic effect; 2. Promote the release of active substances by platelets, enhance platelet aggregation and adhesion, shorten coagulation time, and produce a hemostatic effect; 3. Enhance capillary resistance, reduce capillary permeability, and thus reduce bleeding. |
0 |