Improves the body's metabolism, participates in the metabolism of fat, protein, sugar, nucleic acid and nucleotides, and is also a source of energy in the body

It plays an important role in the metabolism of sugar, fat and protein, and is closely related to the synthesis of acetylcholine, the accumulation of liver glycogen, the reduction of cholesterol and the regulation of blood lipid content.

It can lower blood sugar, inhibit glycogenolysis and glycogenolysis, promote muscle and fat tissue to absorb glucose and amino acids, and promote the decomposition of very low density lipoprotein.

A derivative of methylxanthine, a bronchodilator, relaxes bronchial smooth muscle by inhibiting phosphodiesterase in smooth muscle cells, thereby suppressing asthma.

It works by inhibiting the normal protein synthesis of sensitive microorganisms and has a wide range of antimicrobial effects, mainly targeting Gram-negative rods and a few Gram-positive bacteria, including Citrobacter, Enterobacter, Escherichia coli, Klebsiella, Morgan Proteus, Pseudomonas aeruginosa, Salmonella, Shigella and Staphylococcus (penicillin- and methicillin-resistant). It is also active in vitro against some isolated Acinetobacter, Neisseria, indole-positive Proteus, Pseudomonas, and Serratia. Most streptococci and anaerobic microorganisms such as Clostridium are resistant to aminoglycosides.

This product is a thromboxane synthetase inhibitor that can inhibit the production of TXA2, thus having the effects of anti-platelet aggregation and vasodilation. Animal experiments have shown that intravenous administration can reduce plasma TXB2 levels and the Keto-PGF12/TXB2 ratio, inhibit platelet aggregation caused by different inducers, and prevent cerebral infarction caused by the middle cerebral artery in rats.

Ganciclovir is a guanine antiviral drug. It is a homologue of acyclovir but has a stronger effect. It has a strong inhibitory effect on cytomegalovirus in AIDS patients. After entering the cell, this product is rapidly phosphorylated to form a monophosphate compound, and then converted into a triphosphate compound by the action of cellular kinase. The phosphorylation process in cells infected with cytomegalovirus is faster than that in normal cells. Triphosphates can competitively inhibit DNA polymerase and be incorporated into the DNA of viruses and host cells, thereby inhibiting DNA synthesis. The inhibitory effect on viral DNA polymerase is stronger than that on host cell DNA polymerase.