Unspecified

This product is a non-steroidal antipyretic analgesic with antipyretic, analgesic and anti-inflammatory effects. It has almost no effect on platelet aggregation.

1. Remove lipids and lipoproteins from the blood, remove cholesterol from the blood vessels around the heart, prevent and treat atherosclerosis, and increase the conversion rate of lipids and fatty acids in cells; 2. Effectively prevent and treat coronary heart disease, have anti-atherosclerotic and anti-atherogenic plaque formation effects on experimental atherosclerosis models, increase the coronary artery branches or collateral circulation of atherosclerosis, and accelerate the healing, regeneration and repair of myocardial necrosis or degeneration caused by experimental coronary atherosclerosis or embolism; 3. Increase the biosynthesis of cellular messenger RNA (mRNA) and deoxyribonucleic acid (DNA) and promote cell metabolism; 4. Has a mild anticoagulant effect. Each 1 mg of chondroitin sulfate A is equivalent to the anticoagulant activity of 0.45 U of heparin. This anticoagulant activity is independent of antithrombin III and can exert anticoagulant activity through the fibrinogen system; 5. Has anti-inflammatory, accelerated wound healing and anti-tumor effects.

It is a protein with iron porphyrin as a cofactor that exists in the mitochondria of cells. It is responsible for transferring electrons and is essential for cell respiration. When tissues are hypoxic, exogenous agents can enter the cells and play a role in correcting cell respiration and material metabolism.

Amikacin sulfate is an aminoglycoside antibiotic that has good effects on most Enterobacteriaceae, Pseudomonas aeruginosa and some other Pseudomonas, Acinetobacter, Alcaligenes, etc. It also has good antibacterial effects on meningococci, gonococci, influenza bacilli, Yersinia, Campylobacter fetus, Mycobacterium tuberculosis and some Mycobacterium. Its antibacterial activity is slightly lower than that of gentamicin, but it is stable to the aminoglycoside inactivating enzymes produced by many intestinal Gram-negative bacilli. The mechanism of action is to act on the 30S subunit of the bacterial ribosome and inhibit bacterial protein synthesis. Combination with semi-synthetic penicillins or cephalosporins often achieves a synergistic antibacterial effect.