-
Founded in:
2014-01-26 -
Country:
China -
Address:
No. 1688, Wutong Road, Meihekou City, Jilin Province -
Tax NO.:
91220581088830315W -
Registered Funds:
19.5 million yuan -
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Clindamycin hydrochloride |
Extract from the above information
More
Extract from the above information |
21462-39-5 | 34 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| L-ASPARTIC ACID |
As the precursor of oxaloacetate in the body, it plays an important role in the tricarboxylic acid cycle and participates in the ornithine cycle, combining ammonia and carbon dioxide to form urea. It has a strong affinity for cells and can be used as a carrier of potassium and magnesium ions to help them enter cells, increase the concentration of potassium and magnesium in cells, accelerate the tricarboxylic acid cycle of liver cells, and have a certain effect on improving liver function and reducing serum bilirubin concentration. It is metabolized and excreted from the body through the kidneys.
More
As the precursor of oxaloacetate in the body, it plays an important role in the tricarboxylic acid cycle and participates in the ornithine cycle, combining ammonia and carbon dioxide to form urea. It has a strong affinity for cells and can be used as a carrier of potassium and magnesium ions to help them enter cells, increase the concentration of potassium and magnesium in cells, accelerate the tricarboxylic acid cycle of liver cells, and have a certain effect on improving liver function and reducing serum bilirubin concentration. It is metabolized and excreted from the body through the kidneys. |
79-91mg | 6899-03-2 | 18 |
| Potassium |
As a carrier of potassium ions, it helps them enter cells, increases the concentration of potassium in cells, accelerates the tricarboxylic acid cycle of liver cells, and has a certain effect on improving liver function and reducing serum bilirubin concentration. It is metabolized and excreted from the body through the kidneys.
More
As a carrier of potassium ions, it helps them enter cells, increases the concentration of potassium in cells, accelerates the tricarboxylic acid cycle of liver cells, and has a certain effect on improving liver function and reducing serum bilirubin concentration. It is metabolized and excreted from the body through the kidneys. |
10.6-12.2mg | 0 | |
| Magnesium |
As a carrier of magnesium ions, it helps them enter cells, increases the concentration of magnesium in cells, accelerates the tricarboxylic acid cycle of liver cells, and has a certain effect on improving liver function and reducing serum bilirubin concentration. It is metabolized and excreted from the body through the kidneys.
More
As a carrier of magnesium ions, it helps them enter cells, increases the concentration of magnesium in cells, accelerates the tricarboxylic acid cycle of liver cells, and has a certain effect on improving liver function and reducing serum bilirubin concentration. It is metabolized and excreted from the body through the kidneys. |
3.9-4.5mg | 0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| L-ASPARTIC ACID |
As the precursor of oxaloacetate in the body, it plays an important role in the tricarboxylic acid cycle and participates in the ornithine cycle, combining ammonia and carbon dioxide to form urea. It has a strong affinity for cells and can be used as a carrier of potassium and magnesium ions to help them enter cells, increase the concentration of potassium and magnesium in cells, accelerate the tricarboxylic acid cycle of liver cells, and have a certain effect on improving liver function and reducing serum bilirubin concentration. It is metabolized and excreted from the body through the kidneys.
More
As the precursor of oxaloacetate in the body, it plays an important role in the tricarboxylic acid cycle and participates in the ornithine cycle, combining ammonia and carbon dioxide to form urea. It has a strong affinity for cells and can be used as a carrier of potassium and magnesium ions to help them enter cells, increase the concentration of potassium and magnesium in cells, accelerate the tricarboxylic acid cycle of liver cells, and have a certain effect on improving liver function and reducing serum bilirubin concentration. It is metabolized and excreted from the body through the kidneys. |
79-91mg | 6899-03-2 | 18 |
| Potassium |
As an ion carrier, it enters cells, increases the concentration of potassium and magnesium in cells, accelerates the tricarboxylic acid cycle of liver cells, and has a certain effect on improving liver function and reducing serum bilirubin concentration. It is metabolized and excreted from the body through the kidneys.
More
As an ion carrier, it enters cells, increases the concentration of potassium and magnesium in cells, accelerates the tricarboxylic acid cycle of liver cells, and has a certain effect on improving liver function and reducing serum bilirubin concentration. It is metabolized and excreted from the body through the kidneys. |
10.6-12.2mg | 0 | |
| Magnesium |
As an ion carrier, it enters cells, increases the concentration of potassium and magnesium in cells, accelerates the tricarboxylic acid cycle of liver cells, and has a certain effect on improving liver function and reducing serum bilirubin concentration. It is metabolized and excreted from the body through the kidneys.
More
As an ion carrier, it enters cells, increases the concentration of potassium and magnesium in cells, accelerates the tricarboxylic acid cycle of liver cells, and has a certain effect on improving liver function and reducing serum bilirubin concentration. It is metabolized and excreted from the body through the kidneys. |
3.9-4.5mg | 0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Isosorbide dinitrate |
Directly relaxes smooth muscle, especially vascular smooth muscle; the dilation effect on post-capillary veins is more persistent than that on arteries. It has no obvious direct effect on the myocardium. Due to the dilation of capacitance vessels, the venous return to the heart is reduced, reducing the preload of the heart. At the same time, the peripheral resistance vessels expand, blood pressure drops, the left ventricular ejection resistance is reduced, and the afterload of the heart is reduced. The reduction of the preload and afterload of the heart reduces the oxygen consumption of the myocardium.
More
Directly relaxes smooth muscle, especially vascular smooth muscle; the dilation effect on post-capillary veins is more persistent than that on arteries. It has no obvious direct effect on the myocardium. Due to the dilation of capacitance vessels, the venous return to the heart is reduced, reducing the preload of the heart. At the same time, the peripheral resistance vessels expand, blood pressure drops, the left ventricular ejection resistance is reduced, and the afterload of the heart is reduced. The reduction of the preload and afterload of the heart reduces the oxygen consumption of the myocardium. |
87-33-2 | 23 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Isosorbide dinitrate |
Directly relaxes smooth muscle, especially vascular smooth muscle; the dilation effect on post-capillary veins is more persistent than that on arteries. It has no obvious direct effect on the myocardium. Due to the dilation of capacitance vessels, the venous return to the heart is reduced, reducing the preload of the heart. At the same time, the peripheral resistance vessels expand, blood pressure drops, the left ventricular ejection resistance is reduced, and the afterload of the heart is reduced. The reduction of the preload and afterload of the heart reduces the oxygen consumption of the myocardium.
More
Directly relaxes smooth muscle, especially vascular smooth muscle; the dilation effect on post-capillary veins is more persistent than that on arteries. It has no obvious direct effect on the myocardium. Due to the dilation of capacitance vessels, the venous return to the heart is reduced, reducing the preload of the heart. At the same time, the peripheral resistance vessels expand, blood pressure drops, the left ventricular ejection resistance is reduced, and the afterload of the heart is reduced. The reduction of the preload and afterload of the heart reduces the oxygen consumption of the myocardium. |
87-33-2 | 23 |