After entering the body, this product is converted into glucuronic acid under the action of enzymes and works to reduce the activity of liver amylase, prevent glycogenolysis, increase liver glycogen content, and reduce fat storage. This product can combine with hydroxyl or carboxyl-containing poisons in the liver and intestines to become low-toxic or non-toxic glucuronic acid conjugates and be excreted in the urine.

This product competitively inhibits the action of catecholamines at the adrenaline beta receptor site, mainly by selectively inhibiting cardiac beta 1 receptors, making them in an inhibited state, but it has a weaker effect on beta 2 receptors. 1 Reduces heart rate and cardiac output at rest and during exercise. 2 Reduces systolic blood pressure during exercise. 3 Inhibits tachycardia caused by isoproterenol. 4 Reduces reflex orthostatic tachycardia. 5 In asthmatic patients, metoprolol tartrate reduces FEV1 (forced expiratory volume in 1 second) and FVC (forced vital capacity) significantly less than non-selective beta blockers (such as propranolol).

This product combines with blood ammonia in the body to generate urea and is excreted from the body, which has the effect of reducing blood ammonia and promoting brain metabolism. This product may be a central mediator that can enhance the activity of glucose phosphatase and facilitate the recovery of brain cell function.

In vitro tests of this product have a destructive or inhibitory effect on the morphology or proliferation of cell lines such as liver cancer, esophageal cancer, laryngeal cancer, lung cancer, and cervical cancer. In vivo tests have a certain inhibitory effect on mouse Ehrlich ascites carcinoma, sarcoma-180 and solid liver cancer. It can increase the respiratory control rate and lysosomal enzyme activity of mitochondria in H22 mouse ascites liver cancer cells, interfere with cancer cell division, block in the M phase, and affect its cycle speed. This product can destroy the skeleton of cancer cells (microfilaments, microtubules), affect the ultrastructure of cancer cells, and cause damage to mitochondria, microvilli and plasma membranes. It can inhibit DNA synthesis of cancer cells, but it does not inhibit normal bone marrow cells, and can increase white blood cells.

By inhibiting the effects of phosphodiesterase in smooth muscle cells, it relaxes smooth muscles and thus achieves the effect of suppressing asthma.