It can promote the redox function of cells, make the liver function vigorous, neutralize toxins, promote white blood cell proliferation, and prevent the development of pathogens.

Combined with adenosine triphosphate to form vitamin B1 pyrophosphate (thiamine diphosphate, cocarboxylase), it is a coenzyme necessary for carbohydrate metabolism; it can inhibit the activity of cholinesterase. When it is deficient, the activity of cholinesterase is enhanced, the hydrolysis of acetylcholine is accelerated, causing nerve impulse conduction disorders, affecting gastrointestinal and myocardial function.

It is a precursor of coenzyme A and is an essential substance for many metabolic processes (including carbohydrates, proteins and lipids). It can participate in the synthesis of substances such as steroids, porphyrins, acetylcholine, and maintain normal epithelial function.

Analgesic effect: It is a peripheral analgesic by inhibiting the synthesis of prostaglandins and other substances, and the possibility of central analgesia is not ruled out; Anti-inflammatory effect: It may inhibit the synthesis of prostaglandins or other inflammatory substances, while inhibiting the release of lysosomal enzymes and leukocyte chemotaxis; Antipyretic effect: It may act on the hypothalamic temperature regulation center to increase heat dissipation and have an antipyretic effect; Anti-rheumatic effect: In addition to antipyretic and analgesic effects, it mainly has anti-inflammatory effects; Inhibition of platelet aggregation: It reduces prostaglandin production by inhibiting platelet cyclooxygenase.

It relaxes vascular smooth muscle, releases nitric oxide (NO) through metabolism to generate isosorbide mononitrate, activates guanylate cyclase to increase cyclic guanosine monophosphate (cGMP) in smooth muscle cells, thereby relaxing vascular smooth muscle, dilating peripheral arteries and veins, reducing myocardial oxygen consumption and increasing oxygen supply, and relieving angina pectoris.