This product belongs to the macrolide antibiotics. It has antibacterial activity against Staphylococcus (except methicillin-resistant strains), all groups of streptococci and Gram-positive bacilli. Neisseria, Haemophilus influenzae, Bordetella pertussis, etc. are also sensitive to this product. This product also has antibacterial effects on various anaerobic bacteria except Bacteroides fragilis and Fusobacterium. It also has inhibitory effects on Legionella, Campylobacter fetus, certain spirochetes, Mycoplasma pneumoniae, Rickettsia and Chlamydia. This product is an antibacterial agent, but it also has a bactericidal effect on certain bacteria at high concentrations. It can penetrate the bacterial cell membrane and reversibly bind to the 50S subunit of the bacterial ribosome near the donor site ("P" site), blocking the transfer RNA (t-RNA) from binding to the "P" site, and also blocking the self-acceptor site ("A" site) of the polypeptide chain.

This product is a blood lipid regulating drug of clofibric acid derivatives. It may reduce the formation of triglycerides in the liver by promoting the decomposition of peripheral fat and reducing the liver's uptake of free fatty acids, and inhibit the synthesis of very low-density lipoprotein apolipoprotein to reduce the production of very low-density lipoprotein. This product can reduce blood triglycerides and increase blood high-density lipoprotein concentrations, and slightly reduce blood low-density lipoprotein cholesterol concentrations (in type IV hyperlipoproteinemia, low-density lipoprotein may increase). Long-term studies have shown that this product can reduce the risk of sudden death and myocardial infarction due to severe coronary heart disease.

It has anti-inflammatory, antipyretic and analgesic effects. Its mechanism of action is to reduce the synthesis of prostaglandins by inhibiting cyclooxygenase. It stops the formation of pain nerve impulses in inflammatory tissues and inhibits inflammatory reactions, including inhibiting the chemotaxis of leukocytes and the release of lysosomal enzymes. As for the antipyretic effect, it acts on the hypothalamic temperature regulation center, causing peripheral vasodilation and sweating, which increases heat dissipation. This central antipyretic effect may also be related to the inhibition of prostaglandin synthesis in the hypothalamus.

Antacids, which neutralize stomach acid and protect the ulcer surface

Promote granulation development and mucosal regeneration

Inhibit glandular secretion and relieve pain caused by smooth spasm

This product has central and peripheral antitussive effects, and its antitussive effect strength is about 1/3 of codeine. In addition to having a direct inhibitory effect on the respiratory center of the medulla oblongata, it also has a mild atropine-like effect. It can relax spasmodic bronchial smooth muscles and reduce airway resistance.