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Founded in:
2003-12-24 -
Country:
China -
Address:
Dongwang, Jiefang East Road, Linfen City -
Tax NO.:
91141000757261333L -
Registered Funds:
38 million yuan -
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Indometacin |
This product has anti-inflammatory, antipyretic and analgesic effects. Its mechanism of action is to reduce the synthesis of prostaglandins by inhibiting cyclooxygenase. It stops the formation of pain nerve impulses in inflammatory tissues and inhibits inflammatory reactions, including inhibiting the chemotaxis of leukocytes and the release of lysosomal enzymes. As for the antipyretic effect, it acts on the hypothalamic temperature regulation center, causing peripheral vasodilation and sweating, which increases heat dissipation. This central antipyretic effect may also be related to the inhibition of prostaglandin synthesis in the hypothalamus.
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This product has anti-inflammatory, antipyretic and analgesic effects. Its mechanism of action is to reduce the synthesis of prostaglandins by inhibiting cyclooxygenase. It stops the formation of pain nerve impulses in inflammatory tissues and inhibits inflammatory reactions, including inhibiting the chemotaxis of leukocytes and the release of lysosomal enzymes. As for the antipyretic effect, it acts on the hypothalamic temperature regulation center, causing peripheral vasodilation and sweating, which increases heat dissipation. This central antipyretic effect may also be related to the inhibition of prostaglandin synthesis in the hypothalamus. |
53-86-1 | 23 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Phenytoin sodium |
It is an anti-epileptic drug and anti-arrhythmic drug. The therapeutic dose does not cause sedation and hypnosis. 1. Animal experiments have shown that this product has a selective antagonistic effect on the tonic phase of super-strong electric shock and convulsion, but is ineffective or even aggravates the spasm phase. Therefore, it is effective for grand mal epilepsy, but ineffective for absence seizures. Its anti-epileptic mechanism has not yet been elucidated. It is generally believed that it increases the outflow of sodium ions from cells and reduces the inflow of sodium ions, thereby stabilizing the nerve cell membrane, increasing the excitation threshold, and reducing the spread of high-frequency discharges in the lesion. 2. In addition, this product shortens the action potential interval and effective refractory period, and can also inhibit the inflow of calcium ions, reduce myocardial autonomicity, inhibit the sympathetic center, and have an inhibitory effect on the ectopic rhythm points of the atrium and ventricle, and increase the threshold of atrial fibrillation and ventricular fibrillation. 3. Its cell membrane stabilization effect and reduced synaptic transmission have anti-neuralgia and skeletal muscle relaxation effects. 4. This product can inhibit the synthesis (or) secretion of collagenase by skin fibroblasts. It can also accelerate the metabolism of vitamin D, cause lymphadenopathy, have anti-folate effects, have inhibitory effects on the hematopoietic system, cause allergic reactions, have enzyme-inducing effects, and intravenous administration can dilate peripheral blood vessels.
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It is an anti-epileptic drug and anti-arrhythmic drug. The therapeutic dose does not cause sedation and hypnosis. 1. Animal experiments have shown that this product has a selective antagonistic effect on the tonic phase of super-strong electric shock and convulsion, but is ineffective or even aggravates the spasm phase. Therefore, it is effective for grand mal epilepsy, but ineffective for absence seizures. Its anti-epileptic mechanism has not yet been elucidated. It is generally believed that it increases the outflow of sodium ions from cells and reduces the inflow of sodium ions, thereby stabilizing the nerve cell membrane, increasing the excitation threshold, and reducing the spread of high-frequency discharges in the lesion. 2. In addition, this product shortens the action potential interval and effective refractory period, and can also inhibit the inflow of calcium ions, reduce myocardial autonomicity, inhibit the sympathetic center, and have an inhibitory effect on the ectopic rhythm points of the atrium and ventricle, and increase the threshold of atrial fibrillation and ventricular fibrillation. 3. Its cell membrane stabilization effect and reduced synaptic transmission have anti-neuralgia and skeletal muscle relaxation effects. 4. This product can inhibit the synthesis (or) secretion of collagenase by skin fibroblasts. It can also accelerate the metabolism of vitamin D, cause lymphadenopathy, have anti-folate effects, have inhibitory effects on the hematopoietic system, cause allergic reactions, have enzyme-inducing effects, and intravenous administration can dilate peripheral blood vessels. |
630-93-3 | 14 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| L-Cystine |
It can promote the redox function of cells, make the liver function vigorous, neutralize toxins, promote white blood cell proliferation, and prevent the development of pathogens.
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It can promote the redox function of cells, make the liver function vigorous, neutralize toxins, promote white blood cell proliferation, and prevent the development of pathogens. |
56-89-3 | 12 | |
| Thiamine chloride |
Combined with adenosine triphosphate to form vitamin B1 pyrophosphate (thiamine diphosphate, cocarboxylase), it is a coenzyme necessary for carbohydrate metabolism; it can inhibit the activity of cholinesterase. When it is deficient, the activity of cholinesterase is enhanced, the hydrolysis of acetylcholine is accelerated, causing nerve impulse conduction disorders, affecting gastrointestinal and myocardial function.
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Combined with adenosine triphosphate to form vitamin B1 pyrophosphate (thiamine diphosphate, cocarboxylase), it is a coenzyme necessary for carbohydrate metabolism; it can inhibit the activity of cholinesterase. When it is deficient, the activity of cholinesterase is enhanced, the hydrolysis of acetylcholine is accelerated, causing nerve impulse conduction disorders, affecting gastrointestinal and myocardial function. |
59-43-8 | 9 | |
| Calcium D-Pantothenate |
It is a precursor of coenzyme A and is an essential substance for many metabolic processes (including carbohydrates, proteins and lipids). It can participate in the synthesis of substances such as steroids, porphyrins, acetylcholine, and maintain normal epithelial function.
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It is a precursor of coenzyme A and is an essential substance for many metabolic processes (including carbohydrates, proteins and lipids). It can participate in the synthesis of substances such as steroids, porphyrins, acetylcholine, and maintain normal epithelial function. |
137-08-6 | 8 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| L-Cystine |
It can promote the redox function of cells, make the liver function vigorous, neutralize toxins, promote white blood cell proliferation, and prevent the development of pathogens.
More
It can promote the redox function of cells, make the liver function vigorous, neutralize toxins, promote white blood cell proliferation, and prevent the development of pathogens. |
56-89-3 | 12 | |
| Thiamine chloride |
Combines with adenosine triphosphate to form vitamin B1 pyrophosphate (thiamine diphosphate, cocarboxylase), which is a coenzyme necessary for carbohydrate metabolism; inhibits the activity of cholinesterase. When it is deficient, the activity of cholinesterase is enhanced, the hydrolysis of acetylcholine is accelerated, causing nerve impulse conduction disorders, affecting gastrointestinal and myocardial function
More
Combines with adenosine triphosphate to form vitamin B1 pyrophosphate (thiamine diphosphate, cocarboxylase), which is a coenzyme necessary for carbohydrate metabolism; inhibits the activity of cholinesterase. When it is deficient, the activity of cholinesterase is enhanced, the hydrolysis of acetylcholine is accelerated, causing nerve impulse conduction disorders, affecting gastrointestinal and myocardial function |
59-43-8 | 9 | |
| Calcium D-Pantothenate |
It is a precursor of coenzyme A and is an essential substance for many metabolic processes (including carbohydrates, proteins and lipids). It can participate in the synthesis of substances such as steroids, porphyrins, acetylcholine, and maintain normal epithelial function.
More
It is a precursor of coenzyme A and is an essential substance for many metabolic processes (including carbohydrates, proteins and lipids). It can participate in the synthesis of substances such as steroids, porphyrins, acetylcholine, and maintain normal epithelial function. |
137-08-6 | 8 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| TETRAHYDROPALMATINE HYDROCHLORIDE |
Non-narcotic analgesics, with analgesic, sedative, sleep-inducing and tranquilizing effects, especially effective for dull pain caused by the gastrointestinal system
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Non-narcotic analgesics, with analgesic, sedative, sleep-inducing and tranquilizing effects, especially effective for dull pain caused by the gastrointestinal system |
6024-85-7 | 1 |