It can be converted into PEG2, PGI2, and PGF2a, and has obvious lipid-lowering and anti-platelet aggregation effects; it has obvious cholesterol-lowering effect, and its efficacy is 163 times that of linoleic acid; it can reduce serum total cholesterol, low-density lipoprotein cholesterol, and very low-density lipoprotein cholesterol in high-fat rats, and significantly increase the level of high-density cholesterol; it can participate in the synthesis and balance of prostaglandins; it can significantly inhibit platelet aggregation and the synthesis of thromboxane A2 (TXA2) by platelets, thereby changing the TXA2/PGI2 ratio, and has the effect of preventing and treating thrombotic cardiovascular diseases; it has a stimulating effect on brown adipose tissue, promotes the activity of brown fatty acid mitochondria, consumes excessive calories and achieves a certain weight loss effect.

It can be converted into PEG2, PGI2, and PGF2a, and has obvious lipid-lowering and anti-platelet aggregation effects; it has obvious cholesterol-lowering effects, and its efficacy is 163 times that of linoleic acid; it can participate in the synthesis and balance of prostaglandins; it can significantly inhibit platelet aggregation and the synthesis of thromboxane A2 (TXA2) by platelets, thereby changing the TXA2/PGI2 ratio, and has the effect of preventing and treating thrombotic cardiovascular diseases; it has a stimulating effect on brown adipose tissue, promotes the activity of brown fatty acid mitochondria, consumes excess calories and achieves a certain weight loss effect.

This product is a nitroimidazole derivative that can inhibit the redox reaction of amoeba and break the nitrogen chain of the protozoa. In vitro tests have shown that when the drug concentration is 1~2mg/L, the histolytica amoeba can undergo morphological changes in 6~20 hours and all are killed within 24 hours. When the concentration is 0.2mg/L, the histolytica amoeba can be killed within 72 hours. This product has a strong effect of killing trichomonas, and its mechanism is unknown. Metronidazole has a killing effect on anaerobic microorganisms, and the metabolites generated when it is reduced in the human body also have anti-anaerobic effects, inhibiting the synthesis of bacterial deoxyribonucleic acid, thereby interfering with the growth and reproduction of bacteria, and ultimately causing bacterial death. It has a carcinogenic effect on some animals.

This product is a non-steroidal anti-inflammatory drug. Its analgesic, anti-inflammatory and antipyretic effects work by inhibiting the synthesis of prostaglandins.

This product is a non-steroidal anti-inflammatory drug. Its analgesic, anti-inflammatory and antipyretic effects work by inhibiting the synthesis of prostaglandins.

This product is a non-steroidal anti-inflammatory drug. Its analgesic, anti-inflammatory and antipyretic effects work by inhibiting the synthesis of prostaglandins.

This product is a non-steroidal anti-inflammatory drug. Its analgesic, anti-inflammatory and antipyretic effects work by inhibiting the synthesis of prostaglandins.