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Jilin Deshang Pharmaceutical Co., Ltd.
  • Founded in:

    2012-06-12
  • Country:

    China China
  • Address:

    North 1st Street, Economic Development Zone, Huinan County, Jilin Province
  • Tax NO.:

    912205235944926971
  • Registered Funds:

    118.7619 million yuan
  • Website:

  • Email:

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Sulfadiazine Ice Yellow Tablets
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Sulfonidine ice yellow tablets

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Vitamin U Belladonna Aluminum Magnesium Capsules
This product is a compound preparation. Each tablet contains 50 mg of vitamin U (iodomethylmethionine), 2.6 mg of belladonna fluid extract, 123 mg of aluminum hydroxide, and 58 mg of magnesium trisilicate.
Name Description Content CAS NO. Registered Holders
Vitamin U (iodomethylmethionine)

Can promote granulation tissue and mucosal regeneration

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Can promote granulation tissue and mucosal regeneration

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Extract Belladonna soft liquid and powder

It can inhibit gastric juice secretion and relieve pain caused by smooth muscle spasm

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It can inhibit gastric juice secretion and relieve pain caused by smooth muscle spasm

2.6mg 0
Aluminum hydroxide

Can neutralize excess stomach acid to relieve symptoms such as stomach pain and heartburn

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Can neutralize excess stomach acid to relieve symptoms such as stomach pain and heartburn

123mg 21645-51-2 14
Magnesium trisilicate

Can neutralize excess stomach acid to relieve symptoms such as stomach pain and heartburn

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Can neutralize excess stomach acid to relieve symptoms such as stomach pain and heartburn

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Nephritis Tablets
Goldenrod, verbena, Imperata root, plantain, gourd shell, white scutellaria.
Name Description Content CAS NO. Registered Holders
Golden flower

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VERBENAE HERBA

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Imperatae Rhizoma

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HERBA PLANTAGINIS

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Gourd Shell

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CYNANCHI STAUNTONII RHIZOMA ET RADIX

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Nitrofurantoin enteric-coated tablets
The main ingredient of this product is nitrofurantoin.
Name Description Content CAS NO. Registered Holders
Nitrofurantoin

This product is an antibacterial drug. Escherichia coli is mostly sensitive to this product, and some strains of Enterobacteriaceae such as Enterobacter aerogenes, Enterobacter cloacae, Proteus, and Klebsiella are sensitive to this product. Aeruginosa is usually resistant to this product. This product has antibacterial effects on Gram-positive bacteria such as Enterococcus. The antibacterial activity of this product is not affected by pus and tissue decomposition products. It is more active in acidic urine. The antibacterial mechanism is to interfere with the oxidoreductase system in the bacteria, thereby blocking its metabolic process.

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This product is an antibacterial drug. Escherichia coli is mostly sensitive to this product, and some strains of Enterobacteriaceae such as Enterobacter aerogenes, Enterobacter cloacae, Proteus, and Klebsiella are sensitive to this product. Aeruginosa is usually resistant to this product. This product has antibacterial effects on Gram-positive bacteria such as Enterococcus. The antibacterial activity of this product is not affected by pus and tissue decomposition products. It is more active in acidic urine. The antibacterial mechanism is to interfere with the oxidoreductase system in the bacteria, thereby blocking its metabolic process.

67-20-9 26
Furosemide Tablets
Main ingredient: Furosemide Chemical name: 2-[(2-furanmethyl)amino]-5-(sulfamoyl)-4-chlorobenzoic acid. Molecular formula: C12H11ClN2O5S Molecular weight: 330.74
Name Description Content CAS NO. Registered Holders
Furosemide

1. Effect on water and electrolyte excretion: It can increase the excretion of water, sodium, chloride, potassium, calcium, magnesium, phosphorus, etc., inhibit the active reabsorption of NaCl by the thick wall segment of the renal tubular loop, reduce the osmotic pressure gradient difference of the medullary interstitial fluid, promote the secretion of K+ by the distal tubule, inhibit the reabsorption of Ca2+ and Mg2+ by the loop of Henle and increase their excretion; short-term medication can increase uric acid excretion, and long-term medication can cause hyperuricemia. 2. Effect on hemodynamics: It can inhibit the activity of prostaglandin degrading enzymes to increase the content of prostaglandin E2, dilate renal blood vessels to reduce renal vascular resistance, and increase renal blood flow; it does not reduce the glomerular filtration rate while diuresis; it can dilate the pulmonary capacitance veins, reduce the permeability of pulmonary capillaries, reduce the amount of blood returning to the heart, and reduce the left ventricular end-diastolic pressure, which is helpful for the treatment of acute left heart failure and adult respiratory distress syndrome.

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1. Effect on water and electrolyte excretion: It can increase the excretion of water, sodium, chloride, potassium, calcium, magnesium, phosphorus, etc., inhibit the active reabsorption of NaCl by the thick wall segment of the renal tubular loop, reduce the osmotic pressure gradient difference of the medullary interstitial fluid, promote the secretion of K+ by the distal tubule, inhibit the reabsorption of Ca2+ and Mg2+ by the loop of Henle and increase their excretion; short-term medication can increase uric acid excretion, and long-term medication can cause hyperuricemia. 2. Effect on hemodynamics: It can inhibit the activity of prostaglandin degrading enzymes to increase the content of prostaglandin E2, dilate renal blood vessels to reduce renal vascular resistance, and increase renal blood flow; it does not reduce the glomerular filtration rate while diuresis; it can dilate the pulmonary capacitance veins, reduce the permeability of pulmonary capillaries, reduce the amount of blood returning to the heart, and reduce the left ventricular end-diastolic pressure, which is helpful for the treatment of acute left heart failure and adult respiratory distress syndrome.

54-31-9 48
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