It has a broad-spectrum antimicrobial effect, including aerobic Gram-negative and Gram-positive bacteria, anaerobes, Rickettsia, spirochetes and Chlamydia. It has a bactericidal effect on Haemophilus influenzae, Streptococcus pneumoniae and Neisseria meningitidis; it only has an antibacterial effect on anaerobic bacteria such as Staphylococcus aureus, Streptococcus pyogenes, Streptococcus viridans, Group B hemolytic Streptococcus, Escherichia coli, Klebsiella pneumoniae, Proteus mirabilis, Salmonella typhi, Salmonella paratyphi, Shigella, Bacteroides fragilis, etc. It is usually resistant to Pseudomonas aeruginosa, Acinetobacter, Enterobacter, Serratia marcescens, indole-positive Proteus, methicillin-resistant Staphylococcus and Enterococcus. It reversibly binds to the 50S subunit of the bacterial ribosome, inhibits protein synthesis, and is completely cross-resistant with chloramphenicol. It has a strong immunosuppressive effect, which is about 6 times stronger than chloramphenicol.

It has antibacterial effects on various Gram-negative bacteria, Gram-positive bacteria and Helicobacter pylori. Its mechanism of action is to inhibit the synthesis of bacterial proteins.

It has a local anesthetic effect. Its mechanism of action is to competitively bind to the nerve membrane lipoprotein, block sodium ions, and hinder the conduction of nerve fibers.

It is a substance necessary for the formation of normal epithelial cells in the digestive tract

The first generation of oral cephalosporin has good antibacterial effect on most strains of penicillinase-producing and non-penicillinase-producing Staphylococcus aureus, coagulase-negative Staphylococcus, Streptococcus pneumoniae, group A hemolytic Streptococcus, etc. It also has certain antibacterial activity against Escherichia coli, Proteus mirabilis, Salmonella, Shigella, Haemophilus influenzae and gonococci. Methicillin-resistant Staphylococcus, Enterococcus, indole-positive Proteus, Enterobacter, Serratia and other Enterobacteriaceae, Pseudomonas aeruginosa and Bacteroides fragilis are resistant to this product.

It has a direct relaxant effect on the smooth muscles of the respiratory tract. Its mechanism of action includes inhibiting phosphodiesterase to increase the intracellular cAMP content, achieving bronchial dilation by releasing endogenous adrenaline and noradrenaline, acting as a purine receptor blocker to counteract the contractile effect of adenine on the respiratory tract, and enhancing the contractility of the diaphragm to improve respiratory function.

It is involved in the formation of antibodies and collagen in the body, tissue repair (including certain redox reactions), the metabolism of phenylalanine, tyrosine, and folic acid, the utilization of iron and carbohydrates, the synthesis of fat and protein, as well as the maintenance of immune function, hydroxylation of 5-hydroxytryptamine, maintaining the integrity of blood vessels, and promoting the absorption of non-heme iron.