It combines with blood ammonia in the body to form urea and is excreted from the body, which has the effect of reducing blood ammonia and promoting brain metabolism. It may be a central mediator that can enhance the activity of glucose phosphatase and facilitate the recovery of brain cell function.

Fluconazole is a new triazole antifungal drug with a broad-spectrum antifungal effect and can selectively inhibit fungal sterol synthesis. It is effective against Candida infection, Cryptococcus neoformans infection, Malassezia saccharolyticus, Microsporum, Trichophyton, Epidermophyton, Blastomyces dermatitidis, Coccidioides immitis, Histoplasma capsulatum, Peyer's chromatin, and Cladosporium carinii. It has a highly selective inhibitory effect on fungal cytochrome p-450-dependent enzymes and has no significant effect on plasma steroid concentrations or adrenocorticotropic hormone effects.

Quinolone antibiotics inhibit bacterial DNA gyrase activity and bacterial DNA replication; they have antibacterial effects on Enterobacteriaceae, Staphylococcus, Streptococcus pneumoniae, Haemophilus influenzae, Pseudomonas aeruginosa, Neisseria gonorrhoeae, Chlamydia, etc.

A highly effective fluorinated and chlorinated corticosteroid with anti-inflammatory, antipruritic and vasoconstrictive effects. It increases the reactivity of β receptors to catecholamine, activates adenylate cyclase to increase CAMP production, and inhibits phosphoethylesterase to reduce CAMP destruction, resulting in an increase in cAMP concentration in cells and inhibiting the release of histamine. Its effect in inhibiting the proliferation of granulomas in rats and mice is similar to that of dexamethasone.

It acts on the second step of folic acid anabolism, selectively inhibits the action of dihydrofolate reductase, and when used in combination with sulfamethoxazole, the bacterial folic acid metabolism is doubly blocked, thereby enhancing the synergistic antibacterial effect and reducing drug-resistant strains.

It acts on dihydrofolate synthase, interfering with the first step of folic acid synthesis. When used in combination with trimethoprim, it double-blocks the bacterial folic acid metabolism, enhances the synergistic antibacterial effect and reduces drug-resistant strains, especially the antibacterial effect on Escherichia coli, Haemophilus influenzae, and Staphylococcus aureus is significantly enhanced compared with a single drug.