Rifaximin is a broad-spectrum intestinal antibiotic that inhibits bacterial RNA synthesis by irreversibly binding to the beta-subunit of bacterial DNA-dependent RNA polymerase, and ultimately inhibits bacterial protein synthesis. It has a bactericidal effect on most Gram-positive and Gram-negative bacteria, including aerobic and anaerobic infections. Since rifaximin is not absorbed by the intestine when taken orally, it exerts its antibacterial effect locally by killing intestinal pathogens.

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It is an orally effective specific angiotensin (AT) II receptor antagonist that selectively acts on the AT1 receptor subtype, does not affect the heart rate, has no inhibitory effect on ACE, and is not likely to cause cough.

It inhibits the co-transport of sodium and chloride ions, affects electrolyte reabsorption, increases the excretion of sodium and chloride, reduces plasma volume, increases plasma renin activity, aldosterone secretion and potassium excretion, and can reduce potassium loss in combination with valsartan.

This product is a whole gastrointestinal tract motility drug, which can enhance esophageal peristalsis and lower esophageal sphincter tension, prevent gastric contents from reflux into the esophagus, and improve esophageal clearance; increase gastric and duodenal contractility and coordination of gastric antrum and duodenum; reduce duodenal and gastric reflux, improve gastric and duodenal emptying; strengthen intestinal movement and promote small and large intestine transit. This product does not increase the basal and pentagastrin-induced gastric acid secretion. In addition, it has almost no effect on plasma prolactin levels.

The anti-ulcer component of licorice can increase the hexosamine content of gastric mucosal cells, improve the defense of gastric mucosa, prolong the life of gastric epithelial cells, and accelerate ulcer healing.

Zinc promotes mucosal regeneration and accelerates ulcer healing; it has a good zinc supplement effect on the overall zinc deficiency model of rats, and long-term use does not cause changes in trace elements in major organs of the body, nor does it cause zinc accumulation; it has a protective and ulcer healing effect on a variety of gastric ulcer models

It has a good zinc supplement effect on the overall zinc deficiency model of rats, and long-term use does not cause changes in trace elements in the main organs of the body, nor does it cause zinc accumulation; it has a certain protective effect and promotes ulcer healing in four models: chronic acetic acid gastric ulcer in rats, stress gastric ulcer in rats, gastric ulcer induced by reserpine in mice, and gastric ulcer in rats caused by pylorus ligation. The anti-ulcer component of licorice can increase the hexosamine component of gastric mucosal cells, improve the defense of gastric mucosa, prolong the life of gastric epithelial cells, and accelerate ulcer healing; zinc also has the effect of promoting mucosal regeneration and accelerating ulcer healing.

The anti-ulcer component of licorice can increase the hexosamine content of gastric mucosal cells, improve the defense of gastric mucosa, prolong the life of gastric epithelial cells, and accelerate ulcer healing.