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Founded in:
2010-01-04 -
Country:
China -
Address:
No. 16 Fengjin Road, Chengyang District, Qingdao City, Shandong Province -
Tax NO.:
91370214697187900G -
Registered Funds:
68.1 million yuan -
Website:
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Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| 6-Methyl-17a-hydroxypregna-4,6-diene-3,20-dione 17-acetate |
This product is a semi-synthetic progesterone derivative, which has a certain inhibitory effect on hormone-dependent tumors. Its mechanism of action is the same as that of medroxyprogesterone, which may be through the influence on the secretion of pituitary gonadotropin, controlling the development and growth of ovarian follicles, thereby reducing the production of estrogen. It acts on androgen receptors, preventing their synthesis and reuse, interfering with their binding with estrogen, and inhibiting tumor cell growth. In addition, it can also antagonize glucocorticoid receptors and interfere with the interaction between steroid hormone receptors and regulatory proteins related to cell growth and differentiation.
More
This product is a semi-synthetic progesterone derivative, which has a certain inhibitory effect on hormone-dependent tumors. Its mechanism of action is the same as that of medroxyprogesterone, which may be through the influence on the secretion of pituitary gonadotropin, controlling the development and growth of ovarian follicles, thereby reducing the production of estrogen. It acts on androgen receptors, preventing their synthesis and reuse, interfering with their binding with estrogen, and inhibiting tumor cell growth. In addition, it can also antagonize glucocorticoid receptors and interfere with the interaction between steroid hormone receptors and regulatory proteins related to cell growth and differentiation. |
0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| 6-Methyl-17a-hydroxypregna-4,6-diene-3,20-dione 17-acetate |
This product is a semi-synthetic progesterone derivative, which has a certain inhibitory effect on hormone-dependent tumors. Its mechanism of action is the same as that of medroxyprogesterone, which may be through the influence on the secretion of pituitary gonadotropin, controlling the development and growth of ovarian follicles, thereby reducing the production of estrogen. It acts on androgen receptors, preventing their synthesis and reuse, interfering with their binding with estrogen, and inhibiting tumor cell growth. In addition, it can also antagonize glucocorticoid receptors and interfere with the interaction between steroid hormone receptors and regulatory proteins related to cell growth and differentiation.
More
This product is a semi-synthetic progesterone derivative, which has a certain inhibitory effect on hormone-dependent tumors. Its mechanism of action is the same as that of medroxyprogesterone, which may be through the influence on the secretion of pituitary gonadotropin, controlling the development and growth of ovarian follicles, thereby reducing the production of estrogen. It acts on androgen receptors, preventing their synthesis and reuse, interfering with their binding with estrogen, and inhibiting tumor cell growth. In addition, it can also antagonize glucocorticoid receptors and interfere with the interaction between steroid hormone receptors and regulatory proteins related to cell growth and differentiation. |
0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Eperisone hydrochloride |
Central skeletal muscle relaxants can inhibit decerebrate rigidity and spinal reflex potentials, reduce muscle spindle sensitivity, have calcium antagonist-like effects and block muscle sympathetic nerves, dilate blood vessels to increase blood flow, and have analgesic effects at the spinal cord level.
More
Central skeletal muscle relaxants can inhibit decerebrate rigidity and spinal reflex potentials, reduce muscle spindle sensitivity, have calcium antagonist-like effects and block muscle sympathetic nerves, dilate blood vessels to increase blood flow, and have analgesic effects at the spinal cord level. |
56839-43-1 | 20 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Eperisone hydrochloride |
It can act on the central nervous system and vascular smooth muscle at the same time, relieve skeletal muscle tension and improve blood flow. It can relieve skeletal muscle tension by improving various muscle tension symptoms, inhibiting spinal reflexes, and acting on the γ-system to reduce the sensitivity of muscle spindles. It can also improve blood flow by dilating blood vessels, thus blocking the vicious cycle of hypertonia → circulatory disorders → muscle pain → hypertonia from multiple aspects.
More
It can act on the central nervous system and vascular smooth muscle at the same time, relieve skeletal muscle tension and improve blood flow. It can relieve skeletal muscle tension by improving various muscle tension symptoms, inhibiting spinal reflexes, and acting on the γ-system to reduce the sensitivity of muscle spindles. It can also improve blood flow by dilating blood vessels, thus blocking the vicious cycle of hypertonia → circulatory disorders → muscle pain → hypertonia from multiple aspects. |
56839-43-1 | 20 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| N-[(S)-1-ethoxycarbonyl-3-phenylpropyl]-L-proline maleate |
This product is an angiotensin converting enzyme inhibitor. After oral administration, it is hydrolyzed into Enalaprilat in the body, which strongly inhibits angiotensin converting enzyme, reduces the content of angiotensin II, causes systemic vasodilation, and causes blood pressure reduction. It has a significant antihypertensive effect on rat models of renal hypertension II, renal hypertension I, and spontaneous hypertension.
More
This product is an angiotensin converting enzyme inhibitor. After oral administration, it is hydrolyzed into Enalaprilat in the body, which strongly inhibits angiotensin converting enzyme, reduces the content of angiotensin II, causes systemic vasodilation, and causes blood pressure reduction. It has a significant antihypertensive effect on rat models of renal hypertension II, renal hypertension I, and spontaneous hypertension. |
0 |