Sodium ions are an important component for maintaining the osmotic pressure and volume of extracellular fluid. Sodium also forms a buffer system in the form of sodium bicarbonate, which plays an important role in regulating the acid-base balance of body fluids. A certain concentration of sodium is a necessary condition for maintaining cell excitability and neuromuscular emergency response.

Potassium is the main intracellular cation and an important component for maintaining intracellular osmotic pressure. Potassium participates in the regulation of acid-base balance by exchanging hydrogen ions outside the cell. Potassium participates in the synthesis of sugar and protein and the energy metabolism of converting adenosine diphosphate into adenosine triphosphate. It also participates in the conduction of nerve impulses and the synthesis of the neurotransmitter acetylcholine at the nerve endings.

Calcium salts in the human body exist in bones to maintain bone hardness. Calcium can reduce the permeability of capillaries, increase the density of capillary walls, reduce exudation, and has anti-inflammatory, detumescent and anti-allergic effects.

Sodium chloride is an electrolyte supplement. Sodium and chloride are important electrolytes in the body, mainly present in extracellular fluid, and play a very important role in maintaining normal blood and extracellular fluid volume and osmotic pressure. The normal serum sodium concentration is 135-145mmol/L, accounting for 92% of plasma cations and 90% of total osmotic pressure, so the amount of plasma sodium plays a decisive role in osmotic pressure. The normal serum chloride concentration is 98-106mmol/L. Sodium and chloride ions in the human body are mainly regulated by the hypothalamus, posterior pituitary and kidneys to maintain the stability of body fluid volume and osmotic pressure.

The final metabolite is sodium bicarbonate, which can correct metabolic acidosis; when hyperkalemia is accompanied by acidosis, it can correct acidosis and allow potassium ions to enter the cells from the blood and extracellular fluid.

Under normal circumstances, human blood also contains a small amount of lactic acid, which is mainly produced by glucose or glycogenolysis, and comes from muscles, skin, brain and cells. After lactic acid is produced, it may be converted into glycogen or acetic acid, or enter the tricarboxylic acid cycle and be decomposed into water and carbon dioxide. Therefore, the final metabolite of sodium lactate is sodium bicarbonate, which can correct metabolic acidosis. When hyperkalemia is accompanied by acidosis, sodium lactate can correct acidosis and allow potassium ions to enter cells from blood and extracellular fluid. The main organs that degrade lactic acid are liver and kidney. When lactic acid metabolism in the body is abnormal or impaired, the therapeutic effect is poor.

When hyperkalemia is accompanied by acidosis, sodium lactate can cause potassium ions to enter cells from the blood and extracellular fluid.

Under normal circumstances, human blood also contains a small amount of lactic acid, which is mainly produced by glucose or glycogenolysis, and comes from muscles, skin, brain and cells. After lactic acid is produced, it may be converted into glycogen or acetic acid, or enter the tricarboxylic acid cycle and be decomposed into water and carbon dioxide. Therefore, the final metabolite of sodium lactate is sodium bicarbonate, which can correct metabolic acidosis. When hyperkalemia is accompanied by acidosis, sodium lactate can correct acidosis and allow potassium ions to enter cells from blood and extracellular fluid. The main organs that degrade lactic acid are liver and kidney. When lactic acid metabolism in the body is abnormal or impaired, the therapeutic effect is poor.

This product is a brain metabolism improving drug, which is a cyclic derivative of г-aminobutyric acid. It has the effect of resisting brain function damage caused by physical and chemical factors. It can promote the generation of ATP in the brain, promote the synthesis of acetylcholine and enhance the conduction of nerve excitement, and has the effect of promoting brain metabolism. It can resist brain function damage caused by physical and chemical factors. It has an improving effect on retrograde amnesia caused by hypoxia. It can enhance memory and improve learning ability.

This product is a brain metabolism improving drug, which is a cyclic derivative of г-aminobutyric acid. It has the effect of resisting brain function damage caused by physical and chemical factors. It can promote the generation of ATP in the brain, promote the synthesis of acetylcholine and enhance the conduction of nerve excitement, and has the effect of promoting brain metabolism. It can resist brain function damage caused by physical and chemical factors. It has an improving effect on retrograde amnesia caused by hypoxia. It can enhance memory and improve learning ability.