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Yantai Luyin Pharmaceutical Co., Ltd.
  • Founded in:

    1993-12-20
  • Country:

    China China
  • Address:

    No. 102, Baishi Road, Yantai City, Shandong Province
  • Tax NO.:

    91370600613415452L
  • Registered Funds:

    132.074 million yuan
  • Website:

  • Email:

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Nitrendipine
Nitrendipine
Name Description Content CAS NO. Registered Holders
Nitrendipine

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Nifedipine
Nifedipine
Name Description Content CAS NO. Registered Holders
Nifedipine

1. This product can simultaneously relax the coronary arteries in the normal blood supply area and the ischemic area, antagonize spontaneous or ergonovine-induced coronary artery spasm, increase the delivery of myocardial oxygen to patients with coronary artery spasm, and relieve and prevent coronary artery spasm. 2. This product can inhibit myocardial contraction, reduce myocardial metabolism, and reduce myocardial oxygen consumption. 3. This product can relax peripheral resistance vessels, reduce peripheral resistance, and can reduce systolic and diastolic blood pressure, reducing cardiac afterload. 4. This product can delay the sinus node function and atrioventricular conduction of the isolated heart; electrophysiological studies of whole animals and humans have not found that this product has the effect of delaying atrioventricular conduction, prolonging the recovery time of the sinus node, and slowing down the sinus node rate.

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1. This product can simultaneously relax the coronary arteries in the normal blood supply area and the ischemic area, antagonize spontaneous or ergonovine-induced coronary artery spasm, increase the delivery of myocardial oxygen to patients with coronary artery spasm, and relieve and prevent coronary artery spasm. 2. This product can inhibit myocardial contraction, reduce myocardial metabolism, and reduce myocardial oxygen consumption. 3. This product can relax peripheral resistance vessels, reduce peripheral resistance, and can reduce systolic and diastolic blood pressure, reducing cardiac afterload. 4. This product can delay the sinus node function and atrioventricular conduction of the isolated heart; electrophysiological studies of whole animals and humans have not found that this product has the effect of delaying atrioventricular conduction, prolonging the recovery time of the sinus node, and slowing down the sinus node rate.

21829-25-4 75
Nifuratel
Name Description Content CAS NO. Registered Holders
Nifuratel

Unspecified

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4936-47-4 13
Xanthinol Nicotinate Injection
Each ampoule contains 300 mg of XanthinolNicotinate
Name Description Content CAS NO. Registered Holders
Xanthinol nicotinate

It reduces peripheral vascular resistance, increases cardiac output, reduces venous pressure, activates fibrinolysis, reduces fibrinogen levels, prevents platelet aggregation, promotes the synthesis of pyrimidine nucleosides (NAD and NADP), increases the concentration of adenine nucleosides (AMP, ADP and ATP), and finally hinders lipolysis, reducing elevated cholesterol and triglyceride levels

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It reduces peripheral vascular resistance, increases cardiac output, reduces venous pressure, activates fibrinolysis, reduces fibrinogen levels, prevents platelet aggregation, promotes the synthesis of pyrimidine nucleosides (NAD and NADP), increases the concentration of adenine nucleosides (AMP, ADP and ATP), and finally hinders lipolysis, reducing elevated cholesterol and triglyceride levels

300mg 437-74-1 9
Scopolamine Butylbromide
Name Description Content CAS NO. Registered Holders
Scopolamine butylbromide

This product is an M cholinergic receptor blocker. Its peripheral effects are similar to those of atropine, with only a slight difference in the degree of action: this product has a stronger antispasmodic effect on smooth muscle than atropine, and can selectively relieve gastrointestinal, bile duct and urinary tract smooth muscle spasms and inhibit their peristalsis. It can also be used to relieve vascular smooth muscle spasms and improve microcirculation; its inhibitory effect on the secretion of glands such as the heart, eye smooth muscle (mydriasis and accommodation paralysis) and salivary glands is weaker than that of atropine. Its central effects mainly include: it has an excitatory effect on the respiratory center; its anti-vertigo and anti-tremor paralysis effects are stronger than atropine; but it has a significant sedative effect on the central nervous system, and a larger dose can produce a hypnotic effect. Therefore, the use of this product rarely causes side effects such as central nervous system excitement, mydriasis, and inhibition of saliva secretion similar to those caused by atropine.

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This product is an M cholinergic receptor blocker. Its peripheral effects are similar to those of atropine, with only a slight difference in the degree of action: this product has a stronger antispasmodic effect on smooth muscle than atropine, and can selectively relieve gastrointestinal, bile duct and urinary tract smooth muscle spasms and inhibit their peristalsis. It can also be used to relieve vascular smooth muscle spasms and improve microcirculation; its inhibitory effect on the secretion of glands such as the heart, eye smooth muscle (mydriasis and accommodation paralysis) and salivary glands is weaker than that of atropine. Its central effects mainly include: it has an excitatory effect on the respiratory center; its anti-vertigo and anti-tremor paralysis effects are stronger than atropine; but it has a significant sedative effect on the central nervous system, and a larger dose can produce a hypnotic effect. Therefore, the use of this product rarely causes side effects such as central nervous system excitement, mydriasis, and inhibition of saliva secretion similar to those caused by atropine.

149-64-4 17
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