Aldosterone antagonist. It can inhibit the sodium retention, potassium excretion and water retention caused by the action of aldosterone on the distal renal tubule. When the blood aldosterone concentration is not high, the effect of this product is also weak. On the contrary, when secondary aldosterone increases, the effect of this drug is significant.

This product is a broad-spectrum antifungal drug that can highly selectively inhibit fungal squalene epoxidase, block the squalene epoxidation reaction during the formation of fungal cell membranes and interfere with the early biosynthesis of fungal sterols, thereby exerting the effect of inhibiting and killing fungi.

Competitive angiotensin converting enzyme inhibitors prevent angiotensin I from converting to angiotensin II, thereby reducing peripheral vascular resistance and reducing water and sodium retention by inhibiting aldosterone secretion. They can also dilate peripheral blood vessels by interfering with the degradation of bradykinin. For patients with heart failure, they can reduce pulmonary capillary wedge pressure and pulmonary vascular resistance, increase cardiac output and exercise tolerance time. They can be secreted through breast milk and can pass through the placenta.

This product is a competitive angiotensin converting enzyme inhibitor, which prevents angiotensin I from converting into angiotensin II, thereby reducing peripheral vascular resistance and reducing water and sodium retention by inhibiting aldosterone secretion. This product can also dilate peripheral blood vessels by interfering with the degradation of bradykinin. For patients with heart failure, this product can also reduce pulmonary capillary wedge pressure and pulmonary vascular resistance, increase cardiac output and exercise tolerance time. This product can be secreted through breast milk and can pass through the placenta.

Antiviral drug. It has an inhibitory effect on herpes simplex virus, varicella zoster virus, cytomegalovirus, etc. in vitro. After entering the cells infected by herpes virus, this product competes with deoxynucleoside for viral thymidine kinase or cell kinase, and the drug is phosphorylated into activated acyclovir triphosphate, and then inhibits viral replication in two ways: ① Interfering with viral DNA polymerase and inhibiting viral replication; ② Under the action of DNA polymerase, it binds to the growing DNA chain, causing the extension of the DNA chain to be interrupted. This product has a special affinity for viruses, but has low toxicity to mammalian host cells.