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This product is a non-peptide endothelin receptor antagonist, which can antagonize endothelin-induced vasoconstriction, blood pressure increase and vascular smooth muscle cell proliferation, reduce vascular endothelial damage; increase NO synthesis, relax vascular smooth muscle; inhibit platelet aggregation, anti-coagulation, and improve blood rheology characteristics. This product can also inhibit cholesterol synthesis, lower blood lipids, scavenge free radicals, prevent lipid peroxidation damage; affect complement, enhance immune function, and have certain analgesic and antispasmodic effects.

This product is a histamine H2 receptor blocker. It has a significant inhibitory effect on gastric acid secretion, and can also inhibit the secretion of pepsin, and has a certain protective effect on animal experimental ulcers. It takes effect about 1 hour after taking the medicine, and the effect can last for more than 12 hours.

Azithromycin is a 15-membered macrolide antibiotic. In vitro tests have shown that azithromycin has antibacterial effects on a variety of common clinical pathogens, including: Gram-positive aerobic bacteria, Staphylococcus aureus, Streptococcus pyogenes (group A beta; hemolytic streptococci), pneumonia (streptococci), a-hemolytic streptococci (grass-green streptococci) and other streptococci, diphtheria (rod-shaped) bacilli. This product shows cross-resistance to erythromycin-resistant Gram-positive bacteria, including fecal streptococci (enterococci) and various methicillin-resistant Staphylococcus strains.

This product is an M cholinergic receptor blocker. Its peripheral effects are similar to those of atropine, with only a slight difference in the degree of action: this product has a stronger antispasmodic effect on smooth muscle than atropine, and can selectively relieve gastrointestinal, bile duct and urinary tract smooth muscle spasms and inhibit their peristalsis. It can also be used to relieve vascular smooth muscle spasms and improve microcirculation; its inhibitory effect on the secretion of glands such as the heart, eye smooth muscle (mydriasis and accommodation paralysis) and salivary glands is weaker than that of atropine. Its central effects mainly include: it has an excitatory effect on the respiratory center; its anti-vertigo and anti-tremor paralysis effects are stronger than atropine; but it has a significant sedative effect on the central nervous system, and a larger dose can produce a hypnotic effect. Therefore, the use of this product rarely causes adverse reactions such as central nervous system excitement, mydriasis, and inhibition of saliva secretion similar to those caused by atropine.