Has central hypotensive effect

Nitroglycerin has the effect of relaxing vascular smooth muscle. Its relaxing effect on post-capillary capacitance vessels and major arteries, especially the coronary arteries that are still responsive, is stronger than its effect on resistance vessels. Systemic vasodilation increases venous blood volume, reduces ventricle volume, filling pressure, and diastolic wall tension (reduced preload). Dilation of the major arteries close to the heart can reduce systemic resistance (reduced afterload) and arterial resistance. The reduction in ventricular radius and decreased ventricular wall tension respectively reduce myocardial oxygen consumption. The reduction in cardiac filling pressure increases blood flow in the subintimal layer of the heart wall that is sensitive to ischemia, thereby increasing myocardial oxygen supply. At the molecular level, nitroglycerin may act through the same mechanism as endothelium-derived relaxing factor (EDRF), that is, by increasing the concentration of cyclic guanosine monophosphate (CGMP).

Extracted from eggs

It participates in the body's fat metabolism, lowers cholesterol, and excites cholinergic neurons. It is the raw material for synthesizing lipoproteins, can transport fat from the liver to the outside of the liver, participates in fat metabolism, and has an effect on the prevention and treatment of fatty liver; it can also lower serum cholesterol and triglycerides, increase high-density lipoprotein and reduce low-density lipoprotein, and has an effect on the prevention and treatment of atherosclerosis; as a precursor of acetylcholine, it can increase the production of acetylcholine in the brain, excite cholinergic neurons, and have an effect on the recovery of brain cell function.