It selectively relaxes the ballistic smooth muscle and directly inhibits the sphincter of Oddi in the biliary tract, thereby relaxing the biliary sphincter and reducing the resistance to passage through the common bile duct and the duodenum. It also reduces the pressure inside the gallbladder and bile duct, promotes the discharge of bile and pancreatic juice, improves appetite, and eliminates abdominal distension. It has antispasmodic, analgesic and choleretic effects.

This product has both histamine H1 receptor antagonism and the effect of inhibiting the release of allergic reaction mediators. It not only has a strong anti-allergic effect, but also has a longer duration of efficacy. Therefore, it is more effective in preventing various bronchial asthma attacks and exogenous asthma than endogenous asthma.

This product is a new type of non-narcotic analgesic, with mild antipyretic and muscle relaxant effects. Its chemical structure belongs to cyclized o-methylbenzylamine. Therefore, it does not have the characteristics of non-steroidal anti-inflammatory drugs, nor is it an opioid receptor agonist. It is effective for moderate and severe pain. An intramuscular injection of 20 mg of this product is equivalent to the effect of 12 mg of morphine. It has a mild respiratory depressant effect. It has no depressant effect on the circulatory system. There is no tolerance and dependence.

It has a good antibacterial effect on human tuberculosis bacteria, and its bactericidal effect is strongest at pH 5-5.5. It is currently the best bactericidal drug for tuberculosis bacteria that grow slowly in phagocytes in an acidic environment. The antibacterial concentration in vivo is 12.5μg/ml, and 50μg/ml can kill tuberculosis bacteria. The concentration that inhibits tuberculosis bacteria inside cells is 10 times lower than that outside cells, and it has almost no antibacterial effect in neutral and alkaline environments. The mechanism of action may be related to pyrazinoic acid, which interferes with dehydrogenase by replacing nicotinamide, prevents dehydrogenation, hinders the use of oxygen by tuberculosis bacteria, affects the normal metabolism of bacteria, and causes death.