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Founded in:
2001-10-31 -
Country:
China -
Address:
No. 39 Fuyang Road, Rizhao City -
Tax NO.:
91371102732618201L -
Registered Funds:
36 million yuan -
Website:
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Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Cytidine 5'-Diphosphocholine |
Strengthen the function of the brainstem reticular formation, especially the ascending reticular formation excitatory system which is closely related to consciousness; strengthen the function of the vertebral system and improve motor paralysis; improve cerebral circulation, promote brain metabolism by reducing cerebral blood flow resistance and increasing cerebral blood flow, and play a certain role in promoting brain function recovery and awakening.
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Strengthen the function of the brainstem reticular formation, especially the ascending reticular formation excitatory system which is closely related to consciousness; strengthen the function of the vertebral system and improve motor paralysis; improve cerebral circulation, promote brain metabolism by reducing cerebral blood flow resistance and increasing cerebral blood flow, and play a certain role in promoting brain function recovery and awakening. |
987-78-0 | 3 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Metronidazole |
Metronidazole has a strong antibacterial effect on most anaerobic bacteria, but has no effect on aerobic bacteria and facultative anaerobes. The antibacterial spectrum includes Bacteroides fragilis and other Bacteroides, Fusobacterium, Clostridium perfringens, Eubacterium, Veillonella, Peptococcus and Peptostreptococcus. Actinomycetes, Lactobacillus and Propionibacterium are resistant to this product. Its bactericidal concentration is slightly higher than the inhibitory concentration. This product can also inhibit the redox reaction of amoeba and break the nitrogen chain of the protozoa. In vitro tests have shown that when the drug concentration is 1-2 mg/L, the histolytica can undergo morphological changes in 6-20 hours and all are killed within 24 hours. When the concentration is 0.2 mg/L, the histolytica can be killed within 72 hours. This product has a strong effect of killing trichomonas, and its mechanism is unknown. It has a carcinogenic effect on some animals.
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Metronidazole has a strong antibacterial effect on most anaerobic bacteria, but has no effect on aerobic bacteria and facultative anaerobes. The antibacterial spectrum includes Bacteroides fragilis and other Bacteroides, Fusobacterium, Clostridium perfringens, Eubacterium, Veillonella, Peptococcus and Peptostreptococcus. Actinomycetes, Lactobacillus and Propionibacterium are resistant to this product. Its bactericidal concentration is slightly higher than the inhibitory concentration. This product can also inhibit the redox reaction of amoeba and break the nitrogen chain of the protozoa. In vitro tests have shown that when the drug concentration is 1-2 mg/L, the histolytica can undergo morphological changes in 6-20 hours and all are killed within 24 hours. When the concentration is 0.2 mg/L, the histolytica can be killed within 72 hours. This product has a strong effect of killing trichomonas, and its mechanism is unknown. It has a carcinogenic effect on some animals. |
443-48-1 | 39 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Glycerol Fructose |
Glycerol fructose injection is a hyperosmotic preparation that can reduce brain water content and intracranial pressure through hyperosmotic dehydration. This product has a slow onset and a longer duration of action in reducing intracranial pressure.
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Glycerol fructose injection is a hyperosmotic preparation that can reduce brain water content and intracranial pressure through hyperosmotic dehydration. This product has a slow onset and a longer duration of action in reducing intracranial pressure. |
0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Sodium lactate |
The final metabolite is sodium bicarbonate, which can correct metabolic acidosis; when hyperkalemia is accompanied by acidosis, it can correct acidosis and allow potassium ions to enter the cells from the blood and extracellular fluid.
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The final metabolite is sodium bicarbonate, which can correct metabolic acidosis; when hyperkalemia is accompanied by acidosis, it can correct acidosis and allow potassium ions to enter the cells from the blood and extracellular fluid. |
3.10g | 312-85-6 | 0 |
| Sodium chloride |
Under normal circumstances, human blood also contains a small amount of lactic acid, which is mainly produced by glucose or glycogenolysis, and comes from muscles, skin, brain and cells. After lactic acid is produced, it may be converted into glycogen or acetic acid, or enter the tricarboxylic acid cycle and be decomposed into water and carbon dioxide. Therefore, the final metabolite of sodium lactate is sodium bicarbonate, which can correct metabolic acidosis. When hyperkalemia is accompanied by acidosis, sodium lactate can correct acidosis and allow potassium ions to enter cells from blood and extracellular fluid. The main organs that degrade lactic acid are liver and kidney. When lactic acid metabolism in the body is abnormal or impaired, the therapeutic effect is poor.
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Under normal circumstances, human blood also contains a small amount of lactic acid, which is mainly produced by glucose or glycogenolysis, and comes from muscles, skin, brain and cells. After lactic acid is produced, it may be converted into glycogen or acetic acid, or enter the tricarboxylic acid cycle and be decomposed into water and carbon dioxide. Therefore, the final metabolite of sodium lactate is sodium bicarbonate, which can correct metabolic acidosis. When hyperkalemia is accompanied by acidosis, sodium lactate can correct acidosis and allow potassium ions to enter cells from blood and extracellular fluid. The main organs that degrade lactic acid are liver and kidney. When lactic acid metabolism in the body is abnormal or impaired, the therapeutic effect is poor. |
6.00g | 7647-14-5 | 43 |
| Potassium chloride |
When hyperkalemia is accompanied by acidosis, sodium lactate can cause potassium ions to enter cells from the blood and extracellular fluid.
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When hyperkalemia is accompanied by acidosis, sodium lactate can cause potassium ions to enter cells from the blood and extracellular fluid. |
0.30g | 7447-40-7 | 38 |
| Calcium chloride |
Under normal circumstances, human blood also contains a small amount of lactic acid, which is mainly produced by glucose or glycogenolysis, and comes from muscles, skin, brain and cells. After lactic acid is produced, it may be converted into glycogen or acetic acid, or enter the tricarboxylic acid cycle and be decomposed into water and carbon dioxide. Therefore, the final metabolite of sodium lactate is sodium bicarbonate, which can correct metabolic acidosis. When hyperkalemia is accompanied by acidosis, sodium lactate can correct acidosis and allow potassium ions to enter cells from blood and extracellular fluid. The main organs that degrade lactic acid are liver and kidney. When lactic acid metabolism in the body is abnormal or impaired, the therapeutic effect is poor.
More
Under normal circumstances, human blood also contains a small amount of lactic acid, which is mainly produced by glucose or glycogenolysis, and comes from muscles, skin, brain and cells. After lactic acid is produced, it may be converted into glycogen or acetic acid, or enter the tricarboxylic acid cycle and be decomposed into water and carbon dioxide. Therefore, the final metabolite of sodium lactate is sodium bicarbonate, which can correct metabolic acidosis. When hyperkalemia is accompanied by acidosis, sodium lactate can correct acidosis and allow potassium ions to enter cells from blood and extracellular fluid. The main organs that degrade lactic acid are liver and kidney. When lactic acid metabolism in the body is abnormal or impaired, the therapeutic effect is poor. |
0.2g | 10043-52-4 | 27 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Fluconazole |
Fluconazole is a triazole broad-spectrum antifungal drug that selectively inhibits the C-14-alpha;-demethylation of fungal cytochrome P-450 sterols, causing the accumulation of 14-alpha;-methyl sterols in fungi, thereby inhibiting the reproduction and growth of fungi. It has antibacterial activity against Cryptococcus neoformans and Candida species, and is effective against Candida species infections, Cryptococcus neoformans infections, Microsporum and Trichophyton infections, Blastomyces dermatitidis infections, and Coccidioides immitis infections.
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Fluconazole is a triazole broad-spectrum antifungal drug that selectively inhibits the C-14-alpha;-demethylation of fungal cytochrome P-450 sterols, causing the accumulation of 14-alpha;-methyl sterols in fungi, thereby inhibiting the reproduction and growth of fungi. It has antibacterial activity against Cryptococcus neoformans and Candida species, and is effective against Candida species infections, Cryptococcus neoformans infections, Microsporum and Trichophyton infections, Blastomyces dermatitidis infections, and Coccidioides immitis infections. |
86386-73-4 | 69 |