Participates in bone formation and reconstruction of bone tissue after fracture, as well as muscle contraction, nerve transmission, coagulation mechanism and reduces capillary permeability, etc.

It can be converted into PEG2, PGI2, and PGF2a, and has obvious lipid-lowering and anti-platelet aggregation effects; it has obvious cholesterol-lowering effect, and its efficacy is 163 times that of linoleic acid; it can reduce the serum total cholesterol, low-density lipoprotein cholesterol, and very low-density lipoprotein cholesterol in high-fat rats, and significantly increase the level of high-density cholesterol; it can participate in the synthesis and balance of prostaglandins; it can also significantly inhibit platelet aggregation and the synthesis of thromboxane A2 (TXA2) by platelets, thereby changing the TXA2/PGI2 ratio, and has the effect of preventing and treating thrombotic cardiovascular diseases; it has a stimulating effect on brown adipose tissue, promotes the activity of brown fatty acid mitochondria, consumes excessive calories and achieves a certain weight loss effect.

It can be converted into PEG2, PGI2, and PGF2a, and has obvious lipid-lowering and anti-platelet aggregation effects; it has obvious cholesterol-lowering effects, and its efficacy is 163 times that of linoleic acid; it can participate in the synthesis and balance of prostaglandins; it can significantly inhibit platelet aggregation and the synthesis of thromboxane A2 (TXA2) by platelets, thereby changing the TXA2/PGI2 ratio, and has the effect of preventing and treating thrombotic cardiovascular diseases; it has a stimulating effect on brown adipose tissue, promotes the activity of brown fatty acid mitochondria, consumes excess calories and achieves a certain weight loss effect.

It has the effect of competitively blocking the binding of histamine to H2 receptors. It inhibits the effect of gastric acid, which is 5 to 12 times that of cimetidine in terms of moles. Therefore, it is a potent H2 receptor blocker

Sulfonamide antibiotics used for intestinal infections can enhance their antibacterial activity when used in combination with trimethoprim, and convert the antibacterial effect into a bactericidal effect, thereby reducing the production of drug-resistant strains.

When used in combination with sulfonamides, it can double block bacterial folate anabolism, have a synergistic effect, enhance the antibacterial activity of sulfonamides, convert the antibacterial effect into a bactericidal effect, and reduce the production of drug-resistant strains.