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I. Core Neuroprotection: Blocking apoptosis of brain cells, resisting hypoxia-ischemia damage
Inhibiting excitotoxicity of glutamate, calcium overload, blocking the apoptotic pathway after ischemia/trauma, reducing necrosis of damaged neurons, and reducing the volume of injury lesions and cerebral edema after cerebral infarction and brain trauma. PubMed.
Clearing free radicals in the brain, reducing lactic acid accumulation, optimizing aerobic metabolism of brain cells, significantly enhancing the brain's tolerance to hypoxia, and improving dizziness, lightheadedness, and fatigue caused by insufficient blood supply to the brain.
Down-regulating pro-apoptotic enzymes, maintaining the integrity of neuronal structure in the presence of hypoxia, aging, and trauma, and delaying continuous neuronal loss.
II. Neural Regeneration and Synaptic Remodeling, Enhancing Brain Plasticity (Core Cognitive Mechanism)
Activating Shh and BDNF neurotrophic pathways, promoting the proliferation and differentiation of neural stem cells in the hippocampus, inducing the generation of new neurons (neurogenesis) Cerebrolys... 。
Enhance the density of dendritic spines, extend axonal branches, increase the number of synapses, strengthen the efficiency of neural signal transmission, repair damaged neural networks, and improve the speed of brain information processing.
Promote the synthesis and release of neurotransmitters (acetylcholine, dopamine, 5-hydroxytryptamine), optimize the signal transmission basis for memory, concentration, and logical thinking.
Accelerate myelin repair, protect nerve fiber conduction pathways, and improve the sluggishness and expression blockage caused by white matter damage.
III. Improve memory, cognitive decline, and combat dementia-like diseases
Inhibit the deposition of β-amyloid protein, reduce excessive phosphorylation of tau protein, reduce the formation of characteristic neurotoxic plaques in Alzheimer's disease, and slow down the cognitive decline of Alzheimer's disease. (PubMed)
For vascular dementia and mild cognitive decline: Significantly improve memory, attention, and executive ability, reduce the frequency of forgetfulness, forgetfulness, logical confusion attacks; Clinical double-blind trials show that long-term intervention can stabilize the MOCA and ADAS-cog cognitive scores.
Improve brain fatigue caused by brain aging in middle-aged and elderly people, and long-term staying up late and high pressure: Relieve forgetfulness, thinking block, distraction, and difficulty in reading comprehension.
IV. Functional rehabilitation after stroke and craniocerebral trauma surgery
During the recovery period of stroke: Promote the growth of microvessels in the brain, repair the nerve-vessel unit, improve limb muscle strength, balance and coordination, upper limb activity ability, and reduce the degree of disability, and improve the ability of self-care. Cerebrolys... 。
Improve post-stroke aphasia, speech difficulties, and reading disorders, and reconstruct the neural pathways of the language center; reduce the risk of cognitive regression during the recovery period. Cerebrolys... 。
Concussion, post-cerebral surgery: Accelerate the repair of damaged brain tissue, alleviate post-traumatic headache, memory impairment, apathy, sleep disorders, and slow response. PMC.
V. Anti-inflammatory in the brain, alleviating chronic low-grade inflammation in the central nervous system
Block the NF-κB inflammatory pathway, down-regulate pro-inflammatory factors TNF-α and IL-6 in the brain, reduce persistent chronic inflammation (inflammatory brain aging) induced by aging, brain injury, and metabolic abnormalities.
Relieve persistent headache, emotional irritability, nocturnal light sleep with frequent dreams, and physical tension and discomfort caused by inflammation.
VI. Regulation of emotions, sleep and mental state
Balance neurotransmitters in the limbic brain regions, improve emotions, anxiety, irritability, apathy, and depression tendencies secondary to brain injury and aging.
Optimize neural repair metabolism at night, improve brain fatigue, sleep difficulty, fragmented sleep, and dizziness upon waking up after brain injury; note: Active peptides have a mild central stimulating effect, and evening use may affect sleep. It is recommended to use in the morning.
Relieve mental lethargy and lack of motivation caused by long-term brain overuse and insufficient blood supply to the brain.
Hazard Identification
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Other hazards which do not result in classification
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Handling and Storage
Precautions for safe handling
Handling in a well ventilated place. Wear suitable protective clothing. Avoid contact with skin and eyes. Avoid formation of dust and aerosols. Use non-sparking tools. Prevent fire caused by electrostatic discharge steam.
Conditions for safe storage, including any incompatibilities
Store the container tightly closed in a dry, cool and well-ventilated place. Store apart from foodstuff containers or incompatible materials.