Promote normal protein metabolism in the human body, correct negative nitrogen balance, supplement protein, and accelerate wound healing

Promote normal protein metabolism in the human body, correct negative nitrogen balance, supplement protein, and accelerate wound healing

Promote normal protein metabolism in the human body, correct negative nitrogen balance, supplement protein, and accelerate wound healing

Necessary for amino acid metabolism in trauma patients; promotes normal protein metabolism in the human body, corrects negative nitrogen balance, supplements protein, and accelerates wound healing

Promote normal protein metabolism in the human body, correct negative nitrogen balance, supplement protein, and accelerate wound healing

Promote normal protein metabolism in the human body, correct negative nitrogen balance, supplement protein, and accelerate wound healing

Lomeza is a broad-spectrum quinolone antibiotic that has bactericidal effects on Gram-negative bacteria, Gram-positive bacteria and some anaerobic bacteria. It has good antibacterial effects on methicillin-resistant Staphylococcus aureus, ampicillin-resistant influenza bacilli, pipemidic acid-resistant Escherichia coli and bacteria resistant to other drugs. Its antibacterial mechanism is to inhibit the activity of bacterial DNA helicase, thereby inhibiting bacterial DNA transcription and replication, and has bactericidal effects on a variety of Gram-positive and Gram-negative bacteria.

Lomeza is a broad-spectrum quinolone antibacterial drug that has bactericidal effects on Gram-negative bacteria, Gram-positive bacteria and some anaerobic bacteria. It has good antibacterial effects on methicillin-resistant Staphylococcus aureus, ampicillin-resistant influenza bacilli, pipemidic acid-resistant Escherichia coli and bacteria resistant to other drugs. Its antibacterial mechanism is to inhibit the activity of bacterial DNA helicase, thereby inhibiting bacterial DNA transcription and replication, and has bactericidal effects on a variety of Gram-positive and Gram-negative bacteria.

1. Effects on water and electrolyte excretion: It can increase the excretion of water, sodium, chloride, potassium, calcium, magnesium, phosphorus, etc.; by inhibiting the active reabsorption of NaCl by the thick wall segment of the renal tubular loop, it reduces the osmotic pressure gradient difference of the medullary interstitial fluid, resulting in increased excretion of water, Na+, and Cl-; it promotes the secretion of K+ by the distal tubule; it inhibits the reabsorption of Ca2+ and Mg2+ by the loop of Henle and increases their excretion; short-term medication can increase uric acid excretion, and long-term medication can cause hyperuricemia. 2. Effects on hemodynamics: It can inhibit the activity of prostaglandin degrading enzymes, increase the content of prostaglandin E2, and dilate blood vessels; it can dilate renal blood vessels, reduce renal vascular resistance, and increase renal blood flow; it does not reduce the glomerular filtration rate during the diuretic effect; it can dilate the pulmonary capacitance veins, reduce the permeability of pulmonary capillaries, reduce the amount of blood returning to the heart, and reduce the left ventricular end-diastolic pressure, which is helpful for the treatment of acute left heart failure and adult respiratory distress syndrome.

Adrenal cortex hormone drugs. It has anti-inflammatory, anti-allergic, anti-rheumatic and immunosuppressive effects. Its mechanism of action is: 1. Anti-inflammatory effect: It reduces and prevents the tissue's response to inflammation, thereby reducing the manifestations of inflammation. It can inhibit the accumulation of inflammatory cells, including macrophages and leukocytes, at the site of inflammation, and inhibit phagocytosis, the release of lysosomal enzymes, and the synthesis and release of inflammatory chemical mediators. 2. Immunosuppressive effect: It includes preventing or inhibiting cell-mediated immune responses, delayed allergic reactions, reducing the number of T lymphocytes, monocytes, and eosinophils, reducing the ability of immunoglobulins to bind to cell surface receptors, and inhibiting the synthesis and release of interleukins, thereby reducing the transformation of T lymphocytes into lymphoblasts.