A mild peripheral vasodilator with the pharmacological effects of niacin and inositol, it has a lipid-lowering effect; its vasodilatory effect is milder and more lasting than that of niacin, without the side effects of flushing and stomach discomfort; it selectively dilates blood vessels in diseased areas and sensitive areas stimulated by cold; it has the effects of dissolving blood clots, anti-coagulation, anti-fatty liver, and reducing capillary fragility.

1. Hemostasis: It can significantly promote platelet aggregation, significantly shorten coagulation time, and has a significant contraction effect on rabbit arterial vascular strips. 2. Activating blood circulation and removing blood stasis: It can promote the dissolution of rat venous thrombus and accelerate the blood flow rate of mouse ear microcirculation; Panax notoginseng flavonoids can promote blood circulation, indirectly accelerate the electrophoresis rate of red blood cells, reduce blood viscosity, increase coronary flow, reduce myocardial oxygen consumption, and reduce cholesterol. 3. Panax notoginseng saponins can improve the tolerance of brain cells to acute ischemia, and have a protective effect on the stability of nerve cell membranes and microvascular membranes in ischemic brain tissue; Panax notoginseng saponins can also promote protein synthesis, and have a promoting effect on the synthesis of liver, kidney and serum proteins. 4. Rg1 and Rb1 in Panax notoginseng can promote the acquisition, consolidation and reproduction of memory.

It can directly stimulate adrenaline receptors, and can also indirectly stimulate adrenaline receptors by prompting adrenaline nerve endings to release norepinephrine. It has a stimulating effect on both alpha and beta receptors. It can dilate bronchi and constrict local blood vessels, and its action time is longer; it strengthens myocardial contractility, increases cardiac output, and makes venous return to the heart sufficient; it has a stronger central nervous system stimulating effect than adrenaline.

It has ① antihistamine effect, which can compete with histamine released from tissues for H1 receptors on effector cells, thereby stopping allergic reactions; ② at the same time, it inhibits central nervous system activity and causes sedative and hypnotic effects; ③ it enhances the effect of antitussive drugs.

This product is a broad-spectrum semi-synthetic penicillin with extremely low toxicity. The antibacterial spectrum is similar to that of penicillin, and it has a strong antibacterial effect on grass-green streptococci, hemolytic streptococci, and enterococci. Diphtheria bacillus, anthrax bacillus, Gram-positive anaerobic cocci and bacilli are sensitive to this product; meningococci, pertussis bacilli, and Brucella are also very sensitive. Escherichia coli, Salmonella, Shigella dysenteriae, Proteus, influenza bacilli, pneumococci, gonococci, etc., all have varying degrees of drug resistance because some strains produce beta-lactamase. Among them, the resistance rate of Escherichia coli is as high as 50-60%. Enterobacter bacteria, Pseudomonas aeruginosa, and anaerobic Gram-negative bacilli are resistant to this product. This product is mainly used to treat infectious diseases caused by sensitive Staphylococcus aureus, hemolytic streptococci, gonococci, meningococci and diphtheria Bordetella, pertussis Bordetella, influenza Bordetella, Salmonella, Shigella, etc.; such as respiratory tract infections (pneumonia, acute and chronic bronchitis, whooping cough, etc.), gastroenteritis and digestive tract infections (liver and gallbladder infections, acute and chronic gastroenteritis, bacillary dysentery, typhoid and paratyphoid, etc.), urinary tract infections (gonorrhea, urethritis, cystitis, etc.), soft tissue infections and sepsis, meningitis, endocarditis, etc.

It is a semi-synthetic broad-spectrum penicillin antibiotic that mainly acts on the bacterial reproduction stage by inhibiting the biosynthesis of cell wall mucopolypeptides. It is effective against many Gram-positive and Gram-negative bacteria.

It has a β-lactam structure similar to penicillin and has only weak antibacterial activity itself, but it can inactivate enzymes produced by many bacteria by blocking the active site of β-lactamase, especially for clinically important plasmid-mediated β-lactamase. It can protect amoxicillin from hydrolysis and destruction by β-lactamase, so it is still effective against resistant bacteria that produce β-lactamase.