It produces analgesia by inhibiting the synthesis of prostaglandins in the central nervous system and blocking the impulses of pain nerve endings; the antipyretic effect may be related to the inhibition of prostaglandin synthesis in the hypothalamus, leading to peripheral vasodilation, sweating and heat dissipation.

Small doses act on the higher centers of the cerebral cortex to excite the mind, relieve fatigue, and cause cerebral blood vessels to constrict, relieving headaches caused by cerebral vasodilation.

It competitively blocks histamine (H1) receptors, alleviating allergic symptoms, and also has an anti-M cholinergic receptor effect, drying the nasal mucosa.

It has antipyretic and anti-inflammatory effects.

Adrenal cortex hormone drugs. It has anti-inflammatory, anti-allergic, anti-rheumatic and immunosuppressive effects. Its mechanism of action includes: 1. Anti-inflammatory effect: reduce and prevent tissue response to inflammation, inhibit inflammatory cell aggregation, phagocytosis, lysosomal enzyme release and the synthesis and release of inflammatory chemical mediators; 2. Immunosuppressive effect: prevent or inhibit cell-mediated immune response, reduce the number of T lymphocytes, monocytes, eosinophils, reduce the ability of immunoglobulins to bind to cell surface receptors and inhibit interleukin synthesis and release, thereby reducing the transformation of T lymphocytes into lymphoblasts.

In the liver injury model caused by carbon tetrachloride, this product can reduce the serum transaminase and hydroxyproline content in the liver of animals, alleviate the degree of liver lesions and reduce the serum DHBV-DNA level of ducks infected with hepatitis B virus (HBV).