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1. GLP-1 receptor activation effect

Glucose-dependent promotion of insulin secretion by pancreatic β cells: It takes effect only when blood sugar rises, with a low risk of hypoglycemia;

Inhibits the release of glucagon by pancreatic α cells, reduces hepatic gluconeogenesis and hepatic glucose output, and lowers fasting and post-meal blood sugar;

Slows down the gastric emptying rate, prolongs the retention time of food in the stomach, and enhances satiety;

Acts on the hypothalamic appetite regulation area in the brain, inhibits hunger, and reduces the desire for high-calorie and sweet foods.

2. GIP receptor activation effect

Enhances the pancreatic response to glucose, further improving insulin sensitivity; Reduces visceral fat and liver fat accumulation, and improves systemic metabolic disorders.

3. Glucagon (GCG) receptor activation effect

Increases the body's basal energy expenditure, promotes fat oxidation and decomposition, and increases calorie consumption;

Accelerates liver fatty acid metabolism, reduces liver lipid deposition;

Relying on the hypoglycemic counteracting effect of GLP-1/GIP, activating GCG alone will not cause an increase in blood sugar, achieving a balance between fat burning and blood sugar control.