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I. Skeletal Muscle Repair and Myofiber Regeneration (Core Function)
Activate the proliferation and differentiation of muscle satellite stem cells
In vitro experiments on skeletal muscle cells: Wake up dormant satellite cells, promote the division of stem cells, integrate them into damaged muscle fibers, and fill in the tiny tears caused by exercise, which is the most critical pathway for muscle repair.
Accelerate the repair cycle of injured cells
Significantly reduce muscle cell oxidative damage caused by high-intensity metabolism, reduce the accumulation of lactic acid and inflammatory factors, shorten the recovery period of muscle cells, and be used for in vitro mechanism research on exercise fatigue and muscle injury.
Directly promote local muscle hypertrophy
Only effective on muscle tissues subjected to mechanical stimulation, promote myofibril protein deposition and increase the cross-sectional area of myofibrils; will not indiscriminately stimulate the proliferation of soft tissues throughout the body, and achieve the model observation of local muscle mass increase.
Inhibit the protein degradation pathway of muscle
Down-regulate the ubiquitination protein degradation signal, reduce the loss of muscle amino acids, and maintain muscle reserves in cell models of calorie deficit and aging sarcopenia.
II. Muscle Mitochondria and Energy Metabolism Optimization
Promote the birth of skeletal muscle mitochondria
Up-regulate the PGC-1α mitochondrial synthesis gene, increase the number of mitochondria in muscle cells, improve the efficiency of aerobic energy supply, and improve the endurance reserve of muscle cells.
Stabilize the mitochondrial membrane structure and reduce oxidative apoptosis
Remove reactive oxygen free radicals produced by muscle metabolism, maintain mitochondrial membrane potential, and block the programmed cell death of muscle cells induced by oxidative stress.
Optimize the conversion of sugar-lipid energy supply
Improve the glucose transport efficiency of muscle cell membranes, and promote the oxidation and decomposition of fat in the muscle for energy supply, reducing cell indicators related to exercise fatigue and endurance decline.
III. Anti-inflammation and Relief of Chronic Low-level Muscle Inflammation
Down-regulate inflammatory factors of muscle injury
Inhibit the release of injury-induced inflammatory mediators such as TNF-α and IL-6, reduce the inflammatory response of cells related to continuous muscle pain and edema.
Balance the immune microenvironment of muscle tissue
Reduce the risk of connective tissue hyperplasia and fibrosis caused by excessive immunity, avoid long-term muscle injury leading to muscle stiffness and elasticity decline.
IV. Cartilage and Connective Tissue Auxiliary Repair (In vitro Joint Cells)
Stimulate the synthesis of cartilage matrix
In cartilage cell models, increase the secretion of type II collagen and glycosaminoglycans, reduce the apoptosis of cartilage cells caused by mechanical wear.
Activation of tendon and ligament fibroblasts
Promote the synthesis of tendon fibroprotein, strengthen the toughness of connective tissue, used for basic experiments on exercise strain and soft tissue degeneration.
V. Broad-spectrum Anti-apoptosis Protection of Cells (Multiple Tissue In vitro Samples)
Stabilize the anti-apoptosis pathways of various somatic cell mitochondria, have mild antioxidant protection effects on cardiac muscle, vascular endothelium, and nerve cells;
Reduce cell necrosis caused by hypoxia, toxins, and oxidative stress, mainly used for screening studies of ischemic tissue damage.

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Handling and Storage

Precautions for safe handling

Handling in a well ventilated place. Wear suitable protective clothing. Avoid contact with skin and eyes. Avoid formation of dust and aerosols. Use non-sparking tools. Prevent fire caused by electrostatic discharge steam.

Conditions for safe storage, including any incompatibilities

Store the container tightly closed in a dry, cool and well-ventilated place. Store apart from foodstuff containers or incompatible materials.