It participates in the formation of coenzymes in the body. Insufficient intake can lead to vitamin B1 deficiency. Severe deficiency is called beriberi and peripheral neuritis.

Vitamin B1 is an important component of coenzymes required for sugar metabolism

Vitamin B2 is an important coenzyme component required for tissue respiration. Nicotinamide is a component of coenzymes I and II, and is necessary for lipid metabolism and the oxidation of tissue respiration.

Vitamin B6 is a cofactor for many enzymes and is involved in the metabolism of amino acids and fats.

Nicotinamide is a component of coenzymes I and II, essential for lipid metabolism and oxidation of tissue respiration.

Calcium pantothenate is a component of coenzyme A and is involved in the metabolism of sugar, fat, and protein.

Vitamin B1 is an important component of coenzymes required for sugar metabolism

Vitamin B2 is an important coenzyme component required for tissue respiration

Vitamin B6 is a cofactor for many enzymes and is involved in the metabolism of amino acids and fats.

Nicotinamide is a component of coenzymes I and II, essential for lipid metabolism and oxidation of tissue respiration.

Calcium pantothenate is a component of coenzyme A and is involved in the metabolism of sugar, fat, and protein.

This product has anti-inflammatory, antipyretic and analgesic effects. Its mechanism of action is to reduce the synthesis of prostaglandins by inhibiting cyclooxygenase. It stops the formation of pain nerve impulses in inflammatory tissues and inhibits inflammatory reactions, including inhibiting the chemotaxis of leukocytes and the release of lysosomal enzymes. As for the antipyretic effect, it acts on the hypothalamic temperature regulation center, causing peripheral vasodilation and sweating, which increases heat dissipation. This central antipyretic effect may also be related to the inhibition of prostaglandin synthesis in the hypothalamus.

This product is an anti-epileptic drug. Its main metabolite in the body is phenobarbital, which works together. In vitro electrophysiological experiments show that it opens the chloride ion channels of nerve cells and overpolarizes the cells, which is similar to the effect of γ-aminobutyric acid (GABA). At therapeutic concentrations, it can reduce the excitatory effect of glutamate, strengthen the inhibitory effect of γ-aminobutyric acid, inhibit the monosynaptic and polysynaptic transmission of the central nervous system, lead to a decrease in the excitability of the entire nerve cell, and increase the electrical stimulation threshold of the motor cortex. It can increase the threshold of seizures and inhibit the spread of discharges of epileptic foci.