This product is an amide local anesthetic. After blood absorption or intravenous administration, it has obvious biphasic effects of excitation and inhibition on the central nervous system, and there may be no precursor excitation. When the blood concentration is low, analgesia and sleepiness occur, and the pain threshold increases; with increasing doses, the effect or toxicity increases, and there is an anticonvulsant effect at sub-toxic blood concentrations; convulsions may occur when the blood concentration exceeds 5?gml-1. At low doses, this product can promote the outflow of K in myocardial cells, reduce myocardial autonomy, and have an anti-ventricular arrhythmia effect; at therapeutic doses, it has no obvious effect on the electrical activity of myocardial cells, atrioventricular conduction, and myocardial contraction; further increases in blood concentrations can cause a slowing of cardiac conduction velocity, atrioventricular conduction block, inhibition of myocardial contractility, and a decrease in cardiac output.