This non-sulfonylurea insulin secretagogue specifically binds to the 36KDA protein on the ATP-dependent potassium ion channel outside the insulin cell membrane, closing the potassium channel, depolarizing the cell, opening the calcium channel, and causing calcium ion influx, thus promoting insulin secretion. It works faster than sulfonylureas, so it has a faster effect in lowering blood sugar after a meal.

The third generation of oral cephalosporins kills bacteria by inhibiting bacterial cell wall synthesis. It is stable to most beta-lactamases, and many strains producing penicillinase and cephalosporinase are still sensitive to this product. It has good antibacterial effects on Gram-positive cocci such as pneumococci, Streptococcus pyogenes, and Gram-negative bacilli such as Haemophilus influenzae (including enzyme-producing strains), Moraxella catarrhalis (including enzyme-producing strains), Escherichia coli, Proteus mirabilis, and Neisseria gonorrhoeae (including enzyme-producing strains) in vitro and in vivo. It also has antibacterial activity against Streptococcus pneumoniae, Parainfluenzae, Proteus vulgaris, Klebsiella pneumoniae, Pasteurella multocida, Providencia, Salmonella, Shigella, Serratia marcescens, Citrobacter heteromorphis, and Citrobacter malonate, but its clinical effectiveness has not yet been established. It has poor antibacterial effect on Staphylococcus, and has no antibacterial effect on Pseudomonas aeruginosa, Enterobacter, Bacteroides fragilis, Clostridium, etc.

Antibacterial effect, with a wide range of antibacterial spectrum against Gram-positive and Gram-negative bacteria, especially against Gram-positive bacteria such as Staphylococcus aureus and Streptococcus, it has stronger antibacterial activity than previous oral cephalosporins, and its mode of action is bactericidal. It is stable to beta-lactamase produced by various bacteria, and also has excellent antibacterial activity against beta-lactamase producing bacteria. The mechanism of action is to prevent the synthesis of bacterial cell walls, and its action point varies depending on the strain, and it has a strong affinity for the 1 (1a, 1bs).2.3 position of penicillin binding protein (PBP).