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Founded in:
1994-03-08 -
Country:
China -
Address:
No. 11 Xinghua Road, Xiqing District, Tianjin -
Tax NO.:
91120111600580619K -
Registered Funds:
$154.84 million -
Website:
-
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Lansoprazole |
Under acidic conditions, it transforms into an active structure and combines with the SH group of the proton pump ((HK)-ATPase), thereby inhibiting the activity of the enzyme and inhibiting gastric acid secretion; it can inhibit the (HK)-ATPase activity of microsomes in dog gastric mucosa; it has an inhibitory effect on the acid secretion of dog gastric mucosal parietal cells stimulated by histamine, acetylcholine and gastrin; it can significantly inhibit the acid secretion caused by gastrin or insulin in healthy adults, the nocturnal gastric acid secretion and the 24-hour gastric acid secretion; it has a significant and sustained inhibitory effect on the gastric acid secretion of rats and gastric pouch dogs; it can promote the healing of chronic gastric and duodenal ulcers in rats, and has a significant inhibitory effect on gastric ulcers, duodenal ulcers and reflux esophagitis caused by various reasons.
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Under acidic conditions, it transforms into an active structure and combines with the SH group of the proton pump ((HK)-ATPase), thereby inhibiting the activity of the enzyme and inhibiting gastric acid secretion; it can inhibit the (HK)-ATPase activity of microsomes in dog gastric mucosa; it has an inhibitory effect on the acid secretion of dog gastric mucosal parietal cells stimulated by histamine, acetylcholine and gastrin; it can significantly inhibit the acid secretion caused by gastrin or insulin in healthy adults, the nocturnal gastric acid secretion and the 24-hour gastric acid secretion; it has a significant and sustained inhibitory effect on the gastric acid secretion of rats and gastric pouch dogs; it can promote the healing of chronic gastric and duodenal ulcers in rats, and has a significant inhibitory effect on gastric ulcers, duodenal ulcers and reflux esophagitis caused by various reasons. |
103577-45-3 | 88 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Lansoprazole |
Under acidic conditions, it transforms into an active structure and combines with the SH group of the proton pump ((HK)-ATPase) to inhibit gastric acid secretion; it can inhibit (HK)-ATPase activity, parietal cell acid production, gastric acid secretion caused by various stimuli (such as gastrin, insulin, etc.), nighttime and 24-hour gastric acid secretion; it promotes the healing of chronic ulcers and inhibits ulcer formation; it has a significant inhibitory effect on reflux esophagitis.
More
Under acidic conditions, it transforms into an active structure and combines with the SH group of the proton pump ((HK)-ATPase) to inhibit gastric acid secretion; it can inhibit (HK)-ATPase activity, parietal cell acid production, gastric acid secretion caused by various stimuli (such as gastrin, insulin, etc.), nighttime and 24-hour gastric acid secretion; it promotes the healing of chronic ulcers and inhibits ulcer formation; it has a significant inhibitory effect on reflux esophagitis. |
103577-45-3 | 88 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Voglibose |
Inhibit the activity of α-glucosidase in the intestinal wall cells, delay the degradation of ingested carbohydrates, and thus reduce postprandial blood sugar levels
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Inhibit the activity of α-glucosidase in the intestinal wall cells, delay the degradation of ingested carbohydrates, and thus reduce postprandial blood sugar levels |
83480-29-9 | 11 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Voglibose |
This product is an oral hypoglycemic drug. Its mechanism of hypoglycemic effect is to inhibit the activity of a-glucosidase in the small intestinal wall cells, delay the degradation of ingested carbohydrates, and thus reduce postprandial blood sugar levels.
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This product is an oral hypoglycemic drug. Its mechanism of hypoglycemic effect is to inhibit the activity of a-glucosidase in the small intestinal wall cells, delay the degradation of ingested carbohydrates, and thus reduce postprandial blood sugar levels. |
83480-29-9 | 11 |