Bikang Pharmaceutical Xinyi Group Holdings Co., Ltd.
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Founded in:
2001-06-28 -
Country:
China -
Address:
South of Ma Lingshan West Road and west of Gansu Road, Xinyi Economic and Technological Development Zone -
Tax NO.:
913203817287243869 -
Registered Funds:
348 million yuan -
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Panax notoginsenosides |
It has anti-cerebral ischemia effect, can improve brain edema in rats with multiple cerebral infarction, promote glial cell reaction, accelerate the absorption and organization of softening foci, and reduce neuronal lesions in the hippocampus; it has a protective effect on delayed neuronal damage in the hippocampus of gerbils with transient cerebral ischemia, can reduce the Ca2+ content in brain tissue after ischemia, reduce the number of dead neurons, and increase neuronal density. It has anti-myocardial ischemia effect, can reduce the downward shift of the S-T segment of the electrocardiogram in rats with acute myocardial ischemia, and adjust the electrocardiogram inhomogeneity. It improves blood rheology, reduces whole blood viscosity and plasma viscosity, reduces hematocrit and platelet aggregation rate, slows down erythrocyte sedimentation rate, and reduces fibrinogen content. It improves microcirculation, reduces the morphological integral, flow integral, semi-circular integral, and overall integral of the nail fold microcirculation, reduces peri-loop exudation, reduces tube diameter, increases flow velocity, reduces red blood cell aggregation, reduces stasis at the top of the loop, increases loop clarity, and subsides edema. It has the effect of lowering blood lipids, reducing total cholesterol, triglycerides, and low-density lipoprotein levels. It has the effect of resisting free radical damage, increasing the activity of superoxide dismutase in erythrocytes, reducing the content of plasma malondialdehyde, and reducing the content of serum protein kinase C and aspartate aminotransferase.
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It has anti-cerebral ischemia effect, can improve brain edema in rats with multiple cerebral infarction, promote glial cell reaction, accelerate the absorption and organization of softening foci, and reduce neuronal lesions in the hippocampus; it has a protective effect on delayed neuronal damage in the hippocampus of gerbils with transient cerebral ischemia, can reduce the Ca2+ content in brain tissue after ischemia, reduce the number of dead neurons, and increase neuronal density. It has anti-myocardial ischemia effect, can reduce the downward shift of the S-T segment of the electrocardiogram in rats with acute myocardial ischemia, and adjust the electrocardiogram inhomogeneity. It improves blood rheology, reduces whole blood viscosity and plasma viscosity, reduces hematocrit and platelet aggregation rate, slows down erythrocyte sedimentation rate, and reduces fibrinogen content. It improves microcirculation, reduces the morphological integral, flow integral, semi-circular integral, and overall integral of the nail fold microcirculation, reduces peri-loop exudation, reduces tube diameter, increases flow velocity, reduces red blood cell aggregation, reduces stasis at the top of the loop, increases loop clarity, and subsides edema. It has the effect of lowering blood lipids, reducing total cholesterol, triglycerides, and low-density lipoprotein levels. It has the effect of resisting free radical damage, increasing the activity of superoxide dismutase in erythrocytes, reducing the content of plasma malondialdehyde, and reducing the content of serum protein kinase C and aspartate aminotransferase. |
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| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Panax notoginsenosides |
It has anti-cerebral ischemia effect, improves brain edema in rats with multiple cerebral infarctions, promotes glial cell reaction, accelerates the absorption and organization of softening foci, reduces neuronal lesions in the hippocampus, protects delayed neuronal damage caused by transient cerebral ischemia in gerbils, reduces the Ca2 content in brain tissue after ischemia, reduces the number of dead neurons, and increases neuronal density; it has anti-myocardial ischemia effect, can reduce the downward shift of the S-T segment of the electrocardiogram caused by acute myocardial ischemia in rats, and adjust the electrocardiogram inhomogeneity of patients with angina pectoris due to coronary heart disease; it improves blood rheology, reduces whole blood viscosity, plasma viscosity, hematocrit, platelet aggregation rate, slows down the erythrocyte sedimentation rate, and reduces Reduce fibrinogen content; improve microcirculation, reduce nail fold microcirculation morphological integral, flow integral, semi-circular integral, and overall integral, reduce peri-loop exudation, reduce tube diameter, increase flow velocity, reduce red blood cell aggregation, reduce loop top congestion, increase tube loop clarity, and reduce edema; it has a lipid-lowering effect, reduces total cholesterol and triglycerides in patients with type 2 diabetes, and reduces serum triglycerides, cholesterol, and low-density lipoprotein levels in patients with angina pectoris and cerebral infarction of coronary heart disease; it has an effect of resisting free radical damage, increasing red blood cell superoxide dismutase activity, reducing plasma malondialdehyde content, and decreasing serum protein kinase C and aspartate aminotransferase levels.
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It has anti-cerebral ischemia effect, improves brain edema in rats with multiple cerebral infarctions, promotes glial cell reaction, accelerates the absorption and organization of softening foci, reduces neuronal lesions in the hippocampus, protects delayed neuronal damage caused by transient cerebral ischemia in gerbils, reduces the Ca2 content in brain tissue after ischemia, reduces the number of dead neurons, and increases neuronal density; it has anti-myocardial ischemia effect, can reduce the downward shift of the S-T segment of the electrocardiogram caused by acute myocardial ischemia in rats, and adjust the electrocardiogram inhomogeneity of patients with angina pectoris due to coronary heart disease; it improves blood rheology, reduces whole blood viscosity, plasma viscosity, hematocrit, platelet aggregation rate, slows down the erythrocyte sedimentation rate, and reduces Reduce fibrinogen content; improve microcirculation, reduce nail fold microcirculation morphological integral, flow integral, semi-circular integral, and overall integral, reduce peri-loop exudation, reduce tube diameter, increase flow velocity, reduce red blood cell aggregation, reduce loop top congestion, increase tube loop clarity, and reduce edema; it has a lipid-lowering effect, reduces total cholesterol and triglycerides in patients with type 2 diabetes, and reduces serum triglycerides, cholesterol, and low-density lipoprotein levels in patients with angina pectoris and cerebral infarction of coronary heart disease; it has an effect of resisting free radical damage, increasing red blood cell superoxide dismutase activity, reducing plasma malondialdehyde content, and decreasing serum protein kinase C and aspartate aminotransferase levels. |
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| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| N,N-DIETHYLNICOTINAMIDE |
It selectively excites the medullary respiratory center and can also act on the carotid body and aortic body chemoreceptors to reflexively excite the respiratory center and increase the respiratory center's sensitivity to carbon dioxide, deepening and accelerating breathing. It has a weak excitatory effect on the vasomotor center. Excessive doses can cause convulsions.
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It selectively excites the medullary respiratory center and can also act on the carotid body and aortic body chemoreceptors to reflexively excite the respiratory center and increase the respiratory center's sensitivity to carbon dioxide, deepening and accelerating breathing. It has a weak excitatory effect on the vasomotor center. Excessive doses can cause convulsions. |
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