This product is a glucocorticoid with anti-inflammatory, immunosuppressive, antitoxin and anti-shock effects. (1) Anti-inflammatory effect: It has an effect on inflammation caused by various causes except viruses. Glucocorticoids reduce and prevent tissue response to inflammation, thereby reducing the symptoms of inflammation. They can also inhibit the repair of tissues in the later stages of inflammation and reduce sequelae. (2) Immunosuppressive effect: It prevents or inhibits cell-mediated immune responses, delayed allergic reactions, and reduces the expansion of primary immune responses. (3) Anti-toxic and anti-shock effects: Glucocorticoids can improve the body's tolerance, reduce cell damage, and play a role in protecting the body. It also has the effects of dilating blood vessels, enhancing myocardial contractility, and improving microcirculation.

It has pharmacological effects such as sedation, anticonvulsant, enhancing hypoxia tolerance, and enhancing the body's immune function.

Honey fungus has similar pharmacological effects to Gastrodia elata. Honey fungus fruiting bodies contain mannitol, lecithin, ergosterol, vitamin A (high content), vitamin B, C and various amino acids. It has pharmacological effects such as sedation, anticonvulsant, enhancing hypoxia tolerance, and enhancing the body's immune function.

This product has a broad-spectrum antibacterial effect, especially high antibacterial activity against aerobic Gram-negative bacilli. It has good antibacterial activity in vitro against the following bacteria: most bacteria of the Enterobacteriaceae family, including Citrobacter, Enterobacter cloacae, Enterobacter aerogenes, Escherichia coli, Klebsiella, Proteus, Salmonella, Shigella, Vibrio, Yersinia, etc. It often has antibacterial activity against multi-drug resistant bacteria. It has high antibacterial activity against penicillin-resistant Neisseria gonorrhoeae, enzyme-producing Haemophilus influenzae and Moraxella. It has antibacterial activity against most strains of Pseudomonas such as Pseudomonas aeruginosa. This product has antibacterial activity against methicillin-sensitive Staphylococcus aureus, and only moderate antibacterial activity against Streptococcus pneumoniae, hemolytic Streptococcus and Enterococcus faecalis. It has good antimicrobial activity against Chlamydia trachomatis, Mycoplasma, Legionella, and also has antibacterial activity against Mycobacterium tuberculosis and atypical mycobacteria. The antibacterial activity against anaerobic bacteria is poor. Enoxacin is a bactericide that acts on the A subunit of bacterial DNA helicase, inhibiting DNA synthesis and replication, leading to bacterial death.

This product has a broad-spectrum antibacterial effect, especially high antibacterial activity against aerobic Gram-negative bacilli, and has good antibacterial activity against most bacteria of the Enterobacteriaceae family. It has high antibacterial activity against multidrug-resistant bacteria, penicillin-resistant Neisseria gonorrhoeae, enzyme-producing Haemophilus influenzae, and Moraxella. It has antibacterial activity against most strains of Pseudomonas, such as Pseudomonas aeruginosa. It has antibacterial activity against methicillin-sensitive Staphylococci, and only moderate antibacterial activity against Streptococcus pneumoniae, hemolytic Streptococcus, and Enterococcus faecalis. It has good antimicrobial activity against Chlamydia trachomatis, Mycoplasma, and Legionella, and also has antibacterial activity against Mycobacterium tuberculosis and atypical mycobacteria. It has poor antibacterial activity against anaerobic bacteria. Enoxacin is a bactericide that acts on the A subunit of bacterial DNA helicase, inhibiting DNA synthesis and replication, leading to bacterial death.

1. Effect on water and electrolyte excretion: It can increase the excretion of water, sodium, chloride, potassium, calcium, magnesium, phosphorus, etc., inhibit the active reabsorption of NaCl by the thick wall segment of the renal tubular loop, reduce the osmotic pressure gradient difference of the medullary interstitial fluid, reduce the reabsorption of Na+ and Cl-, promote the excretion of K+ and H+, and increase the excretion of Ca2+ and Mg2+. Short-term medication can increase uric acid excretion, and long-term medication can cause hyperuricemia. 2. Effect on hemodynamics: Furosemide can inhibit the activity of prostaglandin degrading enzymes, increase the content of prostaglandin E2, dilate blood vessels, increase renal blood flow, and reduce renal vascular resistance; while increasing the renal tubular fluid flow, it does not reduce the glomerular filtration rate, which helps prevent acute renal failure; it can also dilate the pulmonary capacitance veins, reduce the permeability of pulmonary capillaries, reduce the amount of blood returning to the heart, and reduce the left ventricular end-diastolic pressure. It is used to treat acute left heart failure and adult respiratory distress syndrome.