This product is a glucocorticoid with anti-inflammatory, immunosuppressive, antitoxin and anti-shock effects. (1) Anti-inflammatory effect: It has an effect on inflammation caused by various causes except viruses. Glucocorticoids reduce and prevent tissue response to inflammation, thereby reducing the symptoms of inflammation. They can also inhibit the repair of tissues in the later stages of inflammation and reduce sequelae. (2) Immunosuppressive effect: It prevents or inhibits cell-mediated immune responses, delayed allergic reactions, and reduces the expansion of primary immune responses. (3) Anti-toxic and anti-shock effects: Glucocorticoids can improve the body's tolerance, reduce cell damage, and play a role in protecting the body. It also has the effects of dilating blood vessels, enhancing myocardial contractility, and improving microcirculation.

It has a strong effect on most Gram-positive and Gram-negative bacteria. It has a good antibacterial effect on Escherichia coli, Klebsiella pneumoniae, Proteus vulgaris, Shigella, and Salmonella, and its activity is slightly worse than kanamycin. It also has a good effect on some Staphylococci, gonococci, and meningococci. It has a weak effect on Streptococcus and Mycobacterium tuberculosis, and is ineffective against Pseudomonas aeruginosa and anaerobic bacteria. This product is cross-resistant with kanamycin. The results of the ototoxicity test showed that the hearing and cochlear pathological damage to guinea pigs was significantly lower than that of kanamycin and gentamicin.

It has local analgesic effect and antiseptic effect

1. Effect on water and electrolyte excretion: It can increase the excretion of water, sodium, chloride, potassium, calcium, magnesium, phosphorus, etc.; mainly by inhibiting the active reabsorption of NaCl by the thick-walled segment of the renal tubular loop, reducing the osmotic pressure gradient difference of the medullary interstitial fluid, leading to increased excretion of water, Na+, and Cl-. 2. Effect on hemodynamics: Furosemide can inhibit the activity of prostaglandin degrading enzymes, increase the content of prostaglandin E2, dilate renal blood vessels, reduce renal vascular resistance, and increase renal blood flow; in addition, it can reduce the permeability of pulmonary capillaries, reduce the amount of blood returning to the heart, and reduce the end-diastolic pressure of the left ventricle, which is helpful for the treatment of acute left heart failure and adult respiratory distress syndrome.