This product is a long-acting non-steroidal anti-inflammatory drug. After entering the body, it is metabolized into diphenyl ethyl ester, which inhibits the activity of cyclooxygenase and reduces the synthesis of prostaglandins. Animal experiments show that the anti-inflammatory and analgesic effects of this product are weaker than indomethacin, but stronger than aspirin.

Unspecified

1. It is a calcium ion antagonist, which exerts its pharmacological effect by regulating the calcium ion influx on the cell membrane of myocardial conduction cells, myocardial contractile cells and arterial smooth muscle cells, but does not change the serum calcium concentration. 2. It dilates the main coronary arteries and arterioles in the normal and ischemic parts of the heart, antagonizes spontaneous or ergonovine-induced coronary artery spasm, increases the myocardial oxygen delivery in patients with coronary artery spasm, relieves and prevents coronary artery spasm; reduces total peripheral resistance, reduces myocardial oxygen consumption, and can be used to treat variant angina and unstable angina. 3. It reduces calcium ion influx, prolongs the effective refractory period of the atrioventricular node, slows down conduction, and can reduce the ventricular rate of patients with chronic atrial fibrillation and atrial flutter; it reduces the frequency of paroxysmal supraventricular tachycardia. 4. It produces a blood pressure-lowering effect by reducing the vascular resistance of the systemic circulation, and generally does not cause postural hypotension or reflex tachycardia. 5. It reduces afterload, inhibits myocardial contraction, and can improve left ventricular diastolic function. 6. Animal experiments suggest that verapamil has a local anesthetic effect that is 1.6 times the molar effect of procaine, but its effect and dosage in humans are still unclear.

Improve microcirculation, increase coronary sinus blood flow, improve hypoxia tolerance, slow down heart rate, and resist ulcers

Improve microcirculation, increase coronary sinus blood flow, improve hypoxia tolerance, slow down heart rate, and resist ulcers

Improve microcirculation, increase coronary sinus blood flow, improve hypoxia tolerance, slow down heart rate, and resist ulcers

Improve microcirculation, increase coronary sinus blood flow, improve hypoxia tolerance, slow down heart rate, and resist ulcers

Improve microcirculation, increase coronary sinus blood flow, improve hypoxia tolerance, slow down heart rate, and resist ulcers

It inhibits the synthesis of bacterial cell wall, has good permeability to the cell wall, is stable to β-lactamase, has a strong affinity for penicillin-binding proteins 1A, 1B and 3, and can strongly inhibit the cross-linking of cell wall mucopeptides.