This drug directly inhibits the Na+-K+ exchange in the distal tubules and collecting ducts of the kidney, thereby increasing the excretion of Na+, C1-, and water, and reducing the excretion of K+.

Iron is a component of hemoglobin in red blood cells. When iron is deficient, the amount of hemoglobin synthesized by red blood cells decreases, causing the red blood cells to become smaller and their oxygen-carrying capacity to decrease, resulting in iron-deficiency anemia. Oral administration of this product can supplement iron and correct iron-deficiency anemia.

It works in the form of lithium ions. Its mechanism of preventing manic attacks is to inhibit the Ca2-dependent release of norepinephrine and dopamine at nerve endings, promote the reuptake of norepinephrine in the synaptic cleft by nerve cells, increase its conversion and inactivation, thereby reducing the concentration of norepinephrine. It can also promote the synthesis and release of serotonin, which helps stabilize mood.

Potassium is the main cation in cells, with a concentration of 150-160mmol/L, while the main cation outside cells is sodium ions, and the serum potassium concentration is only 3.5-5.0mmol/L. The body mainly relies on the Na-K-ATPase on the cell membrane to maintain the concentration difference between K and Na in cells. The acid-base balance in the body has an impact on potassium metabolism. For example, when acidosis occurs, H enters the cell. In order to maintain the potential difference between the inside and outside of the cell, K is released outside the cell, causing or aggravating hyperkalemia. Metabolic disorders can also affect acid-base balance. The normal potassium ion concentration and concentration difference inside and outside the cell are closely related to some important functions of the cell, including maintaining carbohydrate metabolism, glycogen storage, protein metabolism, intracellular osmotic pressure and acid-base balance, and myocardial excitability.