It interferes with the first step of folic acid synthesis. When used in combination with trimethoprim, it can double-block the bacterial folic acid metabolism and has a good antibacterial effect on a variety of bacteria.

It selectively inhibits the action of dihydrofolate reductase, and when used in combination with sulfamethoxazole, it can double-block the bacterial folate metabolism and enhance the synergistic antibacterial effect.

It can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects

It is a central nervous system stimulant that can enhance the antipyretic and analgesic effects of acetaminophen and reduce central nervous system inhibitory effects such as drowsiness and dizziness caused by other drugs.

It is an antihistamine that can relieve runny nose, nasal congestion, and sneezing symptoms.

It has antipyretic and sedative effects

Competitively blocks the binding of histamine to H2 receptors, inhibits gastric acid secretion, and is a potent H2 receptor blocker

It has a killing effect on scabies, bacteria, and fungi, and can remove grease, soften the epidermis, and dissolve keratin.

This product has a killing effect on scabies, bacteria, and fungi, and can remove oil, soften the epidermis, and dissolve keratin. Its mechanism of action is that after sulfur comes into contact with skin and tissue secretions, it produces hydrogen sulfide and pentathionic acid.

This product has a killing effect on scabies, bacteria, and fungi, and can remove oil, soften the epidermis, and dissolve keratin. Its mechanism of action is that after sulfur comes into contact with skin and tissue secretions, it produces hydrogen sulfide and pentathionic acid.

This product has a killing effect on scabies, bacteria, and fungi, and can remove oil, soften the epidermis, and dissolve keratin. Its mechanism of action is that after sulfur comes into contact with skin and tissue secretions, it produces hydrogen sulfide and pentathionic acid.

It has anti-inflammatory, antipyretic and analgesic effects. Its mechanism of action is to reduce the synthesis of prostaglandins by inhibiting cyclooxygenase. It stops the formation of pain nerve impulses in inflammatory tissues and inhibits inflammatory reactions, including inhibiting the chemotaxis of leukocytes and the release of lysosomal enzymes. As for the antipyretic effect, it acts on the hypothalamic temperature regulation center, causing peripheral vasodilation and sweating, which increases heat dissipation. This central antipyretic effect may also be related to the inhibition of prostaglandin synthesis in the hypothalamus.