Like vitamin B1, active thiamine combines with pyrophosphate in the body to form a cocarboxylase, which participates in the oxidative decarboxylation reaction of pyruvate and alpha-ketoglutaric acid in sugar metabolism and is necessary for sugar metabolism. When it is deficient, oxidation is blocked to form pyruvate, lactic acid accumulates, and affects the body's energy supply. Its symptoms are mainly manifested in the nervous and cardiovascular systems, with multiple peripheral neuritis affecting both sensory nerves and motor nerves, manifested as paresthesia, neuralgia, limb weakness, and muscle soreness and atrophy. In terms of cardiovascular system, due to the increase of acetone and lactic acid in the blood, the arterioles are dilated, the diastolic pressure is reduced, and the myocardial metabolism is disturbed, so it is easy to have symptoms of heart failure such as palpitations, shortness of breath, chest tightness, cardiac hypertrophy, liver and lung congestion and peripheral edema. In terms of the digestive tract, it is manifested as decreased appetite.

It can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects

It is an antihistamine that can relieve symptoms such as runny nose, nasal congestion, and sneezing.

Antipyretic and sedative effects

No specific pharmacological effects mentioned

This product is a broad-spectrum anthelmintic. It can block the parasite's uptake of multiple nutrients and glucose, leading to the depletion of glycogen in the parasite, making it impossible for the parasite to survive and reproduce.

This product is a benzimidazole derivative, which is rapidly metabolized into sulfoxide, sulfone alcohol and 2-amine sulfone alcohol in the body. It selectively and irreversibly inhibits the polymerization of the cytoplasmic microtubule system of the parasite's intestinal wall cells, blocks its absorption of various nutrients and glucose, leads to the depletion of endogenous glycogen in the worm, and inhibits the fumarate reductase system, preventing the production of adenosine triphosphate, making it impossible for the worm to survive and reproduce. It can also cause the degeneration of the cytoplasmic microtubules of the worm's intestinal cells, and bind to its microtubule protein, causing intracellular transport blockage, resulting in the accumulation of Golgi endocrine granules, the gradual dissolution of the cytoplasm, and the complete degeneration of the absorbing cells, causing the death of the worm. It has the effect of completely killing hookworm eggs and whipworm eggs and partially killing ascarid eggs. In addition to killing and expelling various nematodes parasitic in animals, it also has a significant killing and expelling effect on tapeworms and cysticerci.