Potassium is the main cation in cells, with a concentration of 150-160 mmol/L, while the main cation outside cells is sodium ions, and the serum potassium concentration is only 3.5-5.0 mmol/L. The body mainly relies on Na+-K+ ATPase on the cell membrane to maintain the difference in K+ and Na+ concentrations inside and outside cells. The acid-base balance in the body has an impact on potassium metabolism. For example, when acidosis occurs, H+ enters the cell. In order to maintain the potential difference inside and outside the cell, K+ is released outside the cell, causing or aggravating hyperkalemia. Metabolic disorders can also affect acid-base balance. The normal potassium ion concentration and concentration difference inside and outside cells are closely related to certain functions of cells, such as carbohydrate metabolism, glycogen storage and protein metabolism, excitability and conductivity of nerves and muscles including myocardium, etc.

After passing through the blood-cerebrospinal fluid barrier, it is decomposed into glutamate and gamma-aminobutyric acid (GABA). Glutamate is involved in the information transmission of the central nervous system. Gamma-aminobutyric acid can antagonize the excitotoxic effects of glutamate, improve nerve cell metabolism, maintain nerve stress resistance, reduce the effect of blood ammonia, and improve brain function.

1 It has a positive inotropic effect on the myocardium, mainly acting on β1 receptors, and has relatively little effect on β2 and α receptors. 2 It can directly excite cardiac β1 receptors to enhance myocardial contraction and increase stroke volume, thereby increasing cardiac output. 3 It can reduce peripheral vascular resistance (reduced afterload), but systolic blood pressure and pulse pressure generally remain unchanged, or only increase due to increased cardiac output. 4 It can reduce ventricular filling pressure and promote atrioventricular node conduction. 5 Myocardial contractility is enhanced, and coronary blood flow and myocardial oxygen consumption often increase. 6 Due to increased cardiac output, renal blood flow and urine volume often increase.

This product is an antifibrinolytic drug that can qualitatively inhibit the binding of plasminogen to fibrin, prevent its activation, and thus inhibit fibrinolysis; high concentrations (100 mg/L) directly inhibit the activity of plasmin and achieve a hemostatic effect.

After passing through the blood-cerebrospinal fluid barrier, it is decomposed into glutamate and gamma-aminobutyric acid (GABA). Glutamate is involved in the information transmission of the central nervous system. Gamma-aminobutyric acid can antagonize the excitotoxic effects of glutamate, improve nerve cell metabolism, maintain nerve stress resistance, reduce the effect of blood ammonia, and improve brain function.