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1. GLP-1 Receptor Activation Effects
Glucose-dependent stimulation of insulin secretion from pancreatic β-cells: effective only when blood glucose levels are elevated, resulting in a low risk of hypoglycemia;
Inhibition of glucagon release from pancreatic α-cells, reducing hepatic gluconeogenesis and hepatic glucose output, thereby lowering fasting and postprandial blood glucose;
Delayed gastric emptying, prolonging the retention time of food in the stomach and enhancing satiety;
Action on central hypothalamic appetite-regulating regions to suppress hunger and reduce cravings for high-calorie and sugary foods.

2. GIP Receptor Activation Effects
Enhanced pancreatic response to glucose, further improving insulin sensitivity; reduction of visceral and liver fat accumulation, ameliorating systemic metabolic disorders.

3. Glucagon (GCG) Receptor Activation Effects
Increased basal energy expenditure, promoting fat oxidation and enhancing calorie burning;
Accelerated fatty acid metabolism in the liver, reducing lipid accumulation;
Leveraging the glucose-lowering counteraction of GLP-1/GIP, activation of GCG alone does not cause hyperglycemia, thus achieving a balanced effect of fat burning and blood sugar control.