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I. Appetite Control, Weight and Body Fat Management (Core Function)
Dual central inhibition of hunger, long-lasting enhancement of satiety
Activating the brain's hypothalamus pro-opiomelanocortin receptor, directly suppressing the hunger signal, significantly reducing the craving for high-calorie sweet foods and fried foods; slowing down the gastric emptying rate, significantly prolonging the duration of satiety after eating, naturally reducing the total calorie intake throughout the day, and improving emotional binge eating and midnight snack cravings PMC.
Prioritize the breakdown of visceral fat, reducing fat loss without losing muscle mass
Targeted reduction of abdominal and visceral white fat accumulation, shrinking the volume of fat cells; not decomposing skeletal muscle protein, reducing fat loss without causing relaxation and fatigue after weight loss; used alone can achieve a 6%–11% weight loss, combined with GLP-1 substances, the weight loss effect is significantly improved.
Breakthrough the weight loss plateau period, improving the metabolism prone to obesity
Down-regulating fat storage-related pathways, improving the overall fat oxidation efficiency, solving problems such as unchanged diet and exercise but weight stagnation, water retention and edema after drinking, and fat accumulation after meals.
II. Blood Sugar Regulation, Improvement of Insulin Resistance
Stabilizing post-meal blood sugar, inhibiting excessive output of liver glycogen
Cooperating with insulin after eating to inhibit the release of glucagon, reducing excessive glucose release by the liver, avoiding a sharp increase in post-meal blood sugar, reducing the level of glycosylated hemoglobin, suitable for people with impaired glucose tolerance and high-carbohydrate diets for regulation.
Enhancing overall insulin sensitivity
Reducing cellular insulin resistance, reducing the long-term high-load secretion pressure of the pancreas; reducing the conversion of sugar into fat storage, alleviating fatigue, dizziness, and anxiety caused by post-meal dizziness and blood sugar fluctuations.
Reducing the risk of hypoglycemia
Stable regulation of blood sugar, not continuously lowering blood sugar when blood sugar is low, compared to single hypoglycemic substances, it can autonomously avoid the risk of dizziness and fatigue caused by hypoglycemia.
III. Liver Metabolism Maintenance, Improvement of Fatty Liver Tendency
Accelerating lipid oxidation metabolism in liver cells, reducing liver fat accumulation, alleviating physical heaviness, post-meal abdominal distension, morning fatigue, and dull yellowish complexion caused by non-alcoholic fatty liver disease.
Reducing chronic low-level inflammation in the liver, reducing the burden of long-term high-fat, late-night eating-induced liver damage.
IV. Systemic Chronic Inflammation Regulation
Down-regulating TNF-α and IL-6 circulating inflammatory factors, improving systemic pain, shoulder and neck stiffness, lower extremity edema, and persistent fatigue caused by obesity and metabolic disorders.
Reducing low-level inflammation in the intestinal mucosa, repairing the intestinal barrier, improving irritable bowel syndrome, sticky stool, and food intolerance.
V. Comprehensive Benefits of Cardiovascular Metabolism
Assisting in lowering systolic blood pressure, improving water and sodium retention in obese individuals, and puffy edema;
Regulating blood lipids, reducing triglycerides and low-density lipoprotein, protecting vascular endothelium, maintaining vascular elasticity, and reducing metabolic-related cardiovascular burden.
VI. Recovery and Gain for Exercise Population
Increasing the proportion of fat energy supply, prolonging aerobic endurance, less likely to feel short of breath or fatigue during exercise;
Reducing the accumulation of systemic inflammation after high-intensity training, shortening the recovery period of delayed muscle soreness; reducing the immune window period after training, reducing the probability of seasonal colds.
VII. Endocrine Auxiliary Regulation
Stabilizing stress hormone cortisol, improving stress-induced binge eating and hormone disorder caused by high pressure and staying up late;
Assisting in improving insulin resistance, abdominal obesity, and sweet food craving associated with polycystic ovary syndrome (only dietary adjustment, cannot replace gynecological drugs).
Important Limitations and Safety Tips
Oral Absorption Shortcomings
The ordinary oral formulation without enteric-coating is completely ineffective, only the laboratory-modified enteric-coated version has a weak activity, the effect is mild, the onset period is 8–12 weeks ; skin application cannot penetrate the barrier, there is no effect of reducing fat loss, anti-aging, or skin repair.
Not a Drug
Only clinical research  peptides, no approved oral treatment formulation, cannot replace regular clinical drugs for obesity, type 2 diabetes, and fatty liver.

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GHS label elements, including precautionary statements

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Handling and Storage

Precautions for safe handling

Handling in a well ventilated place. Wear suitable protective clothing. Avoid contact with skin and eyes. Avoid formation of dust and aerosols. Use non-sparking tools. Prevent fire caused by electrostatic discharge steam.

Conditions for safe storage, including any incompatibilities

Store the container tightly closed in a dry, cool and well-ventilated place. Store apart from foodstuff containers or incompatible materials.