Zincate(1-), [N,N-bis[(carboxy-κO)methyl]glycinato(3-)-κN,κO]-, hydrogen (1:1), (T-4)-

254.5 g/mol

252.956479 g/mol

258-370-6

H92631TZMG

DTXSID90201206

121 Ų

SRP: At the time of review, criteria for land treatment or burial (sanitary landfill) disposal practices are subject to significant revision. Prior to implementing land disposal of waste residue (including waste sludge), consult with environmental regulatory agencies for guidance on acceptable disposal practices.

Zinc and Health. Current Bibliographies in Medicine 98-3. Public Services Division/National Library of Medicine. 3619 citations January, 1990-June, 1998. Available from: http://www.nlm.nih.gov/archive/20040831/pubs/cbm/zinc.html as of November 2, 1998.|USEPA; Ambient Water Quality Criteria Doc: Zinc (1980) EPA 400/5-80-079|USEPA; Ambient Water Quality Criteria Doc: Zinc (1987) EPA 400/5-87-003|Arvidson, B. A Review of Axonal Transport of Metals. Toxicology 88: 1-14 (19994) Recent findings regarding axonal transport of metals, including zinc.|For more Special Reports (Complete) data for ZINC (2+) NTA (6 total), please visit the HSDB record page.

Upon intravenous injection zinc is distributed to erythrocytes, plasma, and leukocytes; about 80% of the zinc present in the blood is found in the erythrocytes, 12-20% in the plasma, and about 3% in the leukocytes. ... There is a dynamic exchange of zinc between plasma and erythrocytes. Inhalation of dusts of zinc salts results in a transient accumulation in the lung before its absorption into the blood. There is little absorption of zinc or its salts through the skin. ... About 80% of parenterally administered zinc is excreted in the feces, suggesting an enterohepatic circulation for zinc. In humans, about 10% of the absorbed zinc is excreted in the urine, and in tropical climates about 2-3 mg zinc/day may be lost in sweat. /Soluble zinc compounds/

... NTA carries zinc into the renal tubular ultrafiltrate, where it is reabsorbed by the tubular epithelium. Zinc is toxic to ... /tubular epithelium/ cells, producing cell injury and death leading to a hyperplastic and eventually, neoplastic response.|When normal stimulated cultured human lymphocytes were pretreated with various soluble zinc salts, there was an increase in the numbers of cells with chromosomal fragmentation, diploidy, dicentrism, and chromatid gaps and breaks. A later study using a similar treatment regimen indicated that zinc induced a dose-dependent decrease in lymphocytic mitotic indices. /Soluble zinc salts/