Silver iodide

234.77

233.809555

232-038-0

DTXSID0064836

Light yellow, powder; crystals are hexagonal or cubic

28432900

0

0.88570

Yellow Solid

5.67 g/cm³

552 °C

1506 °C

H2O: 0.03 mg/L

Store in a tightly closed container. Store in a cool, dry, well-ventilated area away from incompatible substances. Store protected from light.

1 MM HG @ 820 DEG C

Odorless

Tasteless

Slowly attacked by boiling concn acids, not affected by hot soln of alkali hydroxides; slowly darkened by light.|It dissolves in the presence of excess iodide ion forming an AgI-2 complex; silver iodide exists in one of three crystal structures depending on the temp, a phenomenon frequently referred to as trimorphism. Below 137 °C, silver iodide is in the cold cubic, or gamma form; at 137-145.8 °C, it exists in the green-yellow colored hexagonal, or beta form; above 145.8 °C, the yellow cubic or alpha form of silver iodide is the stable crystal structure.|Silver iodide has the broadest wavelength sensitivity in the visible spectrum.

9

UN 3077 9 / PGIII

3

36/37/38-53

22-24/25

VW4450000

Stability Light-sensitive. Incompatible with strong oxidizing agents.

P273-P501

H410

SRP: At the time of review, criteria for land treatment or burial (sanitary landfill) disposal practices are subject to significant revision. Prior to implementing land disposal of waste residue (including waste sludge), consult with environmental regulatory agencies for guidance on acceptable disposal practices.|Precipitation is the preferred treatment process for removing toxic heavy metals from electroplating waters. Precipitation processes include hydroxide, lime and/or sulfide treatment. Chemical reduction is used to treat complex metals such as nickel, copper, hexavalent chromium waste, soluble lead, silver, metal containing cyanide, and mercury. Adsorption has shown potential for treating and polishing aqueous metal bearing wastes. Activated carbon, activated alumina, and iron filings are all applicable adsorbents. Alkaline chlorination and incineration are effective cyanide destruction treatments. Evaporation, ion-exchange, reverse osmosis, electrodialysis, and electrolytic recovery are waste reduction and recovery techniques applicable to metal bearing hazardous streams.

... Silver iodide forms impact sensitive systems with sodium and other alkali metals.|A mixture of potassium and any of the following cmpd produces a weak explosion on impact: ... silver iodide ... .

|Warning|H400 (53.09%): Very toxic to aquatic life [Warning Hazardous to the aquatic environment, acute hazard]|P273, P391, and P501|Aggregated GHS information provided by 83 companies from 5 notifications to the ECHA C&L Inventory. Each notification may be associated with multiple companies.|Not Classified

Wear appropriate clothing to prevent any reasonable probability of skin contact. Wear eye protection to prevent any possibility of eye contact. Employees should wash promptly when skin is wet or contaminated. ...

... Work clothing should be changed daily if it is possible that clothing is contaminated. Remove non-impervious clothing prompty if wet or contaminated. Provide emergency eyewash.|SRP: Contaminated protective clothing should be segregated in such a manner so that there is no direct personal contact by personnel who handle, dispose, or clean the clothing. Quality assurance to ascertain the completeness of the cleaning procedures should be implemented before the decontaminated protective clothing is returned for reuse by the workers.

Permissible Exposure Limit: Table Z-1 8-hr Time Weighted Avg: 0.01 mg/cu m. /Silver, metal and compounds (as Ag)/

The major hazards encountered in the use and handling of silver iodide stem from its toxicologic properties. Toxic by all routes (ie, inhalation, ingestion, and dermal contact), exposure to this odorless, light yellow, crystalline substance may occur from its use in seeding clouds for rain-making, as a photosensitive agent in photography, as a local antiseptic, and as a chemical intermediate. Effects from exposure may include skin rashes, conjunctivitis, argyria (a permanent ashen-gray discoloration of skin, conjunctiva, and internal organs), headache, fever, hypersensitivity, laryngitis, and bronchitis. Exposure should be minimized by engineering controls (eg, local exhaust ventilation, or process enclosure). In activities where over-exposure may occur, workers should wear impervious clothing, gloves, face protection, and a self-contained breathing apparatus. Such clothing and equipment should be removed before leaving the worksite. Skin that becomes contaminated with silver iodide should be promptly washed. Eating and smoking should be prohibited in silver iodide work areas. Silver iodide may form explosive compounds with sodium, potassium, acetylene, ammonia, and hydrogen peroxide. Silver iodide should be stored in cool, dark areas, away from the above materials. Before shipping silver iodide, consult with the regulatory requirements of the US Department of Transportation. For small dry spills of silver iodide, collect the material and deposit in sealed containers for reclamation. Before implementing land disposal of silver iodide waste, consult with environmental regulatory agencies for guidance.

D011; A solid waste containing silver may or may not become characterized as a hazardous waste when subjected to the Toxicity Characteristic Leaching Procedure listed in 40 CFR 261.24, and if so characterized, must be managed as a hazardous waste. /Silver/

The emission of silver iodide crystals during cloud seeding has been estimated to result in a silver concn in air of about 0.1 ng/cu m.

Mutagenic potential of copper compounds and its alteration in case of the interaction with silver compounds were analyzed by use of plant test systems. As test systems, Crepis capillaris L., Tradescantia clone 02, and soybean (Glycine max (L.) Merrill) were used. Mutagenic properties of copper iodide and copper sulfate were not detected. CuI, being not a mutagen by itself, remarkably enhanced mutagenic potential of AgI.

The emission of silver iodide crystals during cloud seeding has been estimated to result in a silver concn in air of about 0.1 ng/cu m.

ECOLOGICAL EFFECTS OF SILVER IODIDE ACCUMULATION IN A SEMIARID GRASSLAND ENVIRONMENT. THE POSSIBLE ECOLOGICAL EFFECTS OF SILVER IODIDE ACCUMULATION IN SOIL WHICH MIGHT RESULT FROM WEATHER MODIFICATION WERE EXAMINED BY USE OF A 2.5 YEAR OLD RANDOMIZED TREATMENT PLOT IN A SEMIARID GRASSLAND TO DEFINE POSSIBLE THRESHOLD SILVER LEVELS WHERE CHANGES IN DECOMPOSER PARAMETERS MIGHT FIRST BE DETECTED. AFTER THREE GROWING SEASONS, SILVER IODIDE PRESENCE AT LEVELS ABOVE THOSE WHICH COULD BE EXPECTED FROM WEATHER MODIFICATION APPEARS TO BE RELATED TO DECREASED SOIL OXYGEN UPTAKE, CARBON DIOXIDE EVOLUTION, AND BACTERIAL GLUCOSE MINERALIZATION, THE MOST SENSITIVE ASSAY EVALUATED TO DATE, IS IN THE RANGE OF 1-2 UG G-1 SILVER. IN THE RANGE OF 0-0.6 UG G-1 ACCUMULATED SILVER, NO SIGNIFICANT CHANGES IN THIS PARAMETER WERE OBSERVED IN RELATION TO IMPOSED SILVER.

D011; A waste containing silver may or may not be characterized as a hazardous waste following testing by the Toxicity Characteristic Leaching Procedure as prescribed by the Resource Conservation and Recovery Act (RCRA) regulations. /Silver/

Silver concn in rainwater as a result of /the emission of silver iodide crystals during cloud seeding/ were estimated to be between 0.04 picograms/ml and 5 ng/ml.

Soln containing 5 to 49% of colloidal silver iodide have been employed in the treatment of infections of the mucous membranes. Ointments, usually containing 5%, have been used in inflammatory conditions of the eye, ear, and nose.|MEDICATION: LOCAL ANTI-INFECTIVE; MEDICATION (VET): IN COLLOIDAL SUSPENSIONS AS LOCAL ANTISEPTIC FOR MUCOUS MEMBRANES

ORAL DOSES OF SILVER AS SILVER IODIDE HAD NO EFFECT ON THE MICROORGANISMS OF THE RABBIT CECUM OR GOAT RUMEN. RABBITS ELIMINATED 99% OF ORAL DOSE WITHIN 3 DAYS AND 100% IN 6.3 DAYS. 8-26% ENTERED CECUM, WHERE IT DID NOT ACCUM & WAS NOT ABSORBED. DIGESTION WAS NOT INHIBITED IN RABBITS AND GOATS FOLLOWING THE INGESTION OF DIETS CONTAINING 4.2 AND 1 PPM SILVER AS SILVER IODIDE, OR 10 AND 100 PPM SILVER AS SILVER NITRATE. THUS, AG PROBABLY WILL NOT ADVERSELY AFFECT THE DIGESTIVE MICROFLORA OF LIVESTOCK OR WILDLIFE.|SILVER IODIDE WAS ADMINISTERED ORALLY TO 15 YEARLING EWES (5/GROUP) AT DOSE LEVELS OF 0.1, 1.0, OR 10 MG/KG PER DAY FOR 86 DAYS WITHOUT CLINICAL SIGNS OF TOXICOSIS. ERYTHROCYTE AND LEUKOCYTE COUNTS, PACKED CELL VOLUMES, HB CONCENTRATIONS, AND CIRCULATING THRYOID HORMONE LEVELS (TRIIODOTHYRONINE AND THYROXINE) WERE NOT ALTERED SIGNIFICANTLY IN AGI-DOSED EWES. SILVER AT DOSE LEVELS OF 1 OR 10 MG AGI/KG BODY WT FOR 86 DAYS WAS ABSORBED FROM THE GASTROINTESTINAL TRACT AND DEPOSITED IN SOFT AND HARD TISSUES OF THE BODY. MAXIMUM SILVER RESIDUE FOUND WAS 17 PPM IN THE LIVER OF A EWE RECEIVING 10 MG/KG PER DAY.

Silver iodide was evaluated for mutagenicity in the Ames/microsome test (strains TA 1535, TA 102, TA 97, and TA 98) and for the ability to induce Sister Chromatid Exchanges (SCE) in human cultured lymphocytes and in P388 lymphocytic leukemia cells cultured in the mouse peritoneal cavity. From the cytogenetic in vitro studies, it was observed that silver iodide, either in acetone solutions or as a suspension with polyacryilamide, scarcely causes a doubling effect on SCEs at nearly toxic concentrations (1 ug/ml). Such a doubling effect by silver iodide on SCEs in P388 leukemia cells in vivo was not achieved even after using 100 ug/g mouse body weight. In the Ames/microsome test actually a doubling effect on revertants was only isolately achieved with 30 ug/ml in TA 102 (S9-) and at 150 ug/ml in TA 97 (S9+) doses, which appear to be nearly toxic for bacteria.

Basic treatment: Establish a patent airway. Suction if necessary. Watch for signs of respiratory insufficiency and assist ventilations if needed. Administer oxygen by nonrebreather mask at 10 to 15 L/min. Monitor for pulmonary edema and treat if necessary ... . Monitor for shock and treat if necessary ... . Anticipate seizures and treat if necessary ... . For eye contamination, flush eyes immediately with water. Irrigate each eye continuously with normal saline during transport ... . Do not use emetics. For ingestion, rinse mouth and administer 5 ml/kg up to 200 ml of water for dilution if the patient can swallow, has a strong gag reflex, and does not drool ... . Cover skin burns with dry sterile dressings after decontamination ... . /Poison A and B/|Advanced treatment: Consider orotracheal or nasotracheal intubation for airway control in the patient who is unconscious, has severe pulmonary edema, or is in respiratory arrest. Positive pressure ventilation techniques with a bag valve mask device may be beneficial. Monitor cardiac rhythm and treat arrhythmias as necessary ... . Start an IV with D5W /SRP: "To keep open", minimal flow rate/. Use lactated Ringer's if signs of hypovolemia are present. Watch for signs of fluid overload. Consider drug therapy for pulmonary edema ... . For hypotension with signs of hypovolemia, administer fluid cautiously. Watch for signs of fluid overload ... . Treat seizures with diazepam (Valium) ... . Use proparacaine hydrochloride to assist eye irrigation ... . /Poison A and B/

Seven cloud seeding operators with extensive exposure to silver iodide knew of no persons who had experienced any ill effects due to silver iodide, despite the fact that their hands may have remained yellowed for weeks.|... DEVELOPMENT /OF ARGYRIA (POISONING BY SILVER OR A SILVER SALT WHICH LEADS TO A PERMANENT ASHEN-GRAY DISCOLORATION OF THE SKIN, CONJUNCTIVA, AND INTERNAL ORGANS)/ FROM INHALATION THROUGH OCCUPATIONAL EXPOSURE APPEARS TO BE VERY SLOW & MAY REQUIRE YEARS.

silver iodide

Colloidal silver iodide: Argenti Iodidum Colloidale. Silver iodide rendered colloidally stable by the presence of gelatin.

Silver iodide (AgI): ACTIVE

Agrochemicals -> Repellants